PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26099856-10	2015	Beta cell-specific survivin-knockout pregnant mice displayed glucose intolerance, attenuated beta cell mass expansion and impaired beta cell proliferation, with significant attenuation in the increased expression of Cdk4/Ccnd1, E2f1, p53 (also known as Trp53) and p21 (also known as Cdkn1a) compared with wild-type controls during pregnancy.	Glucose	61-68	baculoviral IAP repeat-containing 5	Mus musculus	19-27	
22870272-7	2012	However, mice deficient in beta-cell survivin showed a defect in beta-cell mass expansion and proliferation with a marked attenuation in the increase of total islet number, largely due to an impairment in the increase in number of larger islets while sparing the increase in number of small islets in the ligated tail of pancreas, resulting in insufficient insulin secretion and glucose intolerance.	Glucose	379-386	baculoviral IAP repeat-containing 5	Mus musculus	37-45	
20807437-6	2010	RESULTS: In pancreatic beta-cells, treatment with glucose, insulin, or EGF increased survivin protein levels at early time points.	Glucose	50-57	baculoviral IAP repeat-containing 5	Mus musculus	85-93	
19644667-7	2009	Targeted deletion of Survivin in the pancreas resulted in a significant decline in beta cell mass throughout the perinatal period, leading to glucose intolerance in the adult.	Glucose	142-149	baculoviral IAP repeat-containing 5	Mus musculus	21-29