PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 33193839-7 2020 ZEB1-AS1 in lung cells was downregulated by high glucose treatment, and overexpression of ZEB1-AS1 resulted in inhibited lung cancer cell apoptosis and downregulated p53. Glucose 49-56 zinc finger E-box binding homeobox 1 Homo sapiens 0-4 32294702-0 2020 lncRNA ZEB1-AS1 inhibits high glucose-induced EMT and fibrogenesis by regulating the miR-216a-5p/BMP7 axis in diabetic nephropathy. Glucose 30-37 zinc finger E-box binding homeobox 1 Homo sapiens 7-11 32294702-14 2020 lncRNA ZEB1-AS1 inhibited high glucose-induced EMT and fibrogenesis via regulating miR-216a-5p/BMP7 axis in diabetic nephropathy, providing a potential target for DN therapy. Glucose 31-38 zinc finger E-box binding homeobox 1 Homo sapiens 7-11 28412735-4 2017 We further show that ZEB1 induced NNMT, rendered cells resistant to glucose deprivation and recapitulated metabolic adaptations and mesenchymal gene expression observed in glucose-restricted cells. Glucose 68-75 zinc finger E-box binding homeobox 1 Homo sapiens 21-25 28111379-0 2017 Knocking down TCF8 inhibits high glucose- and angiotensin II-induced epithelial to mesenchymal transition in podocytes. Glucose 33-40 zinc finger E-box binding homeobox 1 Homo sapiens 14-18 28111379-6 2017 Moreover, a high concentration of glucose and angiotensin II promoted TCF8 expression. Glucose 34-41 zinc finger E-box binding homeobox 1 Homo sapiens 70-74 28111379-7 2017 Inhibiting TCF8 expression with siRNA reversed EMT in podocytes in the presence of a high concentration of glucose and angiotensin. Glucose 107-114 zinc finger E-box binding homeobox 1 Homo sapiens 11-15 28111379-9 2017 Therefore, glucose and angiotensin II may promote EMT in podocytes via TCF8. Glucose 11-18 zinc finger E-box binding homeobox 1 Homo sapiens 71-75 26264186-5 2015 In addition, inhibition of ZEB1 expression potentiated redifferentiation induced by a combination of soluble factors, as judged by an improved response to glucose stimulation and a 3-fold increase in the fraction of C-peptide-positive cells to 60% of BCD cells. Glucose 155-162 zinc finger E-box binding homeobox 1 Homo sapiens 27-31 33215445-0 2020 NORAD regulates proliferation and apoptosis in cardiomyocytes under high-glucose treatment through miRNA-150-5p/ZEB1 axis. Glucose 73-80 zinc finger E-box binding homeobox 1 Homo sapiens 112-116 31209937-6 2019 Our results demonstrated that network pharmacology could provide a guidance role for traditional Chinese medicine research, and mulberry leaves could be of benefit in preventing high glucose-induced EMT in HK-2 cells, which proved that it was related to the upregulation of miR-302a by targeting ZEB1 and the inhibition of NADPH oxidase/ROS/ERK1/2 pathway. Glucose 183-190 zinc finger E-box binding homeobox 1 Homo sapiens 296-300 29227974-9 2018 In vitro, miR-302a-3p expression in HG group increased at 6 h and ascended to the highest level at 12 and 24 h and then gradually decreased from 48 to 72 h. More interesting, ZEB1 protein expression had an opposite change, which gradually decreased from 6 to 24 h and then gradually increased from 48 to 72 h. Moreover, overexpression of miR-302a-3p suppressed expression of ZEB1 in the post-transcriptional level and reversed high glucose-mediated downregulation of E-cadherin and upregulation of vimentin. Glucose 432-439 zinc finger E-box binding homeobox 1 Homo sapiens 175-179