PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19016655-5	2009	The PDZ-domain protein, GIPC (G(alpha)-interacting protein-interacting protein, C-terminus), bound to Glut1 in part via the Glut1 C-terminal PDZ-binding motif, and we found that GIPC deficiency decreased Glut1 surface levels and glucose uptake.	Glucose	229-236	GIPC PDZ domain containing family member 1	Homo sapiens	24-28	
19016655-5	2009	The PDZ-domain protein, GIPC (G(alpha)-interacting protein-interacting protein, C-terminus), bound to Glut1 in part via the Glut1 C-terminal PDZ-binding motif, and we found that GIPC deficiency decreased Glut1 surface levels and glucose uptake.	Glucose	229-236	GIPC PDZ domain containing family member 1	Homo sapiens	30-78	
19016655-8	2009	These results indicate that the C-terminal PDZ-binding motif of Glut1 plays a key role in growth factor regulation of glucose uptake by both allowing GIPC to promote Glut1 trafficking to the cell surface and protecting intracellular Glut1 from lysosomal degradation after growth factor withdrawal, thus allowing the potential for a rapid return of intracellular Glut1 to the cell surface on restimulation.	Glucose	118-125	GIPC PDZ domain containing family member 1	Homo sapiens	150-154	
34643248-10	2021	On the other hand, BBR decreased the interaction between Galpha-interacting protein-interacting protein at the C-terminus (GIPC) and GLUT1, which suggested that the retention of GLUT1 in the cytoplasm may be achieved by inhibiting the interaction between GLUT1 and GIPC, thereby suppressing the glucose transporter function of GLUT1.	Glucose	295-302	GIPC PDZ domain containing family member 1	Homo sapiens	123-127