PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31229266-7	2019	Genetic silencing of HMGA1 significantly inhibited gastric cancer glycolytic activity as revealed by reduced glucose uptake, lactate release, and extracellular acidification ratio.	Glucose	109-116	high mobility group AT-hook 1	Homo sapiens	21-26	
29052178-3	2018	We have previously shown that the chromatin factor high-mobility group A1 (HMGA1) plays a key role in the transcriptional regulation of glucose-responsive genes, including some that are involved in FoxO1-mediated glucose metabolism.	Glucose	136-143	high mobility group AT-hook 1	Homo sapiens	51-73	
29052178-3	2018	We have previously shown that the chromatin factor high-mobility group A1 (HMGA1) plays a key role in the transcriptional regulation of glucose-responsive genes, including some that are involved in FoxO1-mediated glucose metabolism.	Glucose	136-143	high mobility group AT-hook 1	Homo sapiens	75-80	
29052178-3	2018	We have previously shown that the chromatin factor high-mobility group A1 (HMGA1) plays a key role in the transcriptional regulation of glucose-responsive genes, including some that are involved in FoxO1-mediated glucose metabolism.	Glucose	213-220	high mobility group AT-hook 1	Homo sapiens	51-73	
29052178-3	2018	We have previously shown that the chromatin factor high-mobility group A1 (HMGA1) plays a key role in the transcriptional regulation of glucose-responsive genes, including some that are involved in FoxO1-mediated glucose metabolism.	Glucose	213-220	high mobility group AT-hook 1	Homo sapiens	75-80	
22706202-8	2012	Ectopic expression of HMGA1 increased glucose uptake, whereas its knockdown caused AMPK activation.	Glucose	38-45	high mobility group AT-hook 1	Homo sapiens	22-27	
22706202-10	2012	Together, these data show that elevated CAV1 upregulates glucose uptake and ATP production through HMGA1-mediated GLUT3 transcription, suggesting that CAV1 may render tumor cells growth advantages by enhancing aerobic glycolysis.	Glucose	57-64	high mobility group AT-hook 1	Homo sapiens	99-104	
30034366-6	2018	Moreover, other observations have indicated the role of HMGA1 as a positive modulator of the Forkhead box protein O1 (FoxO1), a master regulatory factor for gluconeogenesis and glycogenolysis, as well as a positive regulator of the expression of insulin and of a series of circulating proteins that are involved in glucose counterregulation, such as the insulin growth factor binding protein 1 (IGFBP1), and the retinol binding protein 4 (RBP4).	Glucose	315-322	high mobility group AT-hook 1	Homo sapiens	56-61	
30034366-7	2018	Thus, several lines of evidence underscore the importance of HMGA1 in the regulation of glucose production and disposal.	Glucose	88-95	high mobility group AT-hook 1	Homo sapiens	61-66