PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 17460268-3 2007 RESULTS: Treatment of HTM cells with cytochalasin D and latrunculin A led to significant activation of MMP-2, p38 MAPK, and ERK1/2, which appeared to correlate with changes in cell morphology and actin depolymerization. Cytochalasin D 37-51 mitogen-activated protein kinase 14 Homo sapiens 110-118 15137059-8 2004 Cytochalasin D also activated p38 MAP kinase, which pathway contributed to the cytochalasin D-induced increase in IL-8 production. Cytochalasin D 0-14 mitogen-activated protein kinase 14 Homo sapiens 30-33 16979163-3 2006 We demonstrate that vinblastin and cytochalasin D, which respectively disorganize microtubules and actin microfilaments, trigger the activation of the p38/MAPKAP2 kinase pathway and lead to the specific alphaB-crystallin phosphorylation at serine 59. Cytochalasin D 35-49 mitogen-activated protein kinase 14 Homo sapiens 151-154 16153249-4 2005 Rickettsia inactivation by heat or formaldehyde abolished the activation of p38 kinase and inhibition of cellular invasion by infection at low temperature, pre-treatment of host EC with cytochalasin D, or pre-incubation of rickettsiae with an irreversible phospholipase inhibitor led to a diminished p38 phosphorylation, suggesting requirement of invasion by viable rickettsiae for this host cell response. Cytochalasin D 186-200 mitogen-activated protein kinase 14 Homo sapiens 76-79 15137059-8 2004 Cytochalasin D also activated p38 MAP kinase, which pathway contributed to the cytochalasin D-induced increase in IL-8 production. Cytochalasin D 79-93 mitogen-activated protein kinase 14 Homo sapiens 30-33