PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17460268-3	2007	RESULTS: Treatment of HTM cells with cytochalasin D and latrunculin A led to significant activation of MMP-2, p38 MAPK, and ERK1/2, which appeared to correlate with changes in cell morphology and actin depolymerization.	Cytochalasin D	37-51	mitogen-activated protein kinase 14	Homo sapiens	110-118	
16153249-4	2005	Rickettsia inactivation by heat or formaldehyde abolished the activation of p38 kinase and inhibition of cellular invasion by infection at low temperature, pre-treatment of host EC with cytochalasin D, or pre-incubation of rickettsiae with an irreversible phospholipase inhibitor led to a diminished p38 phosphorylation, suggesting requirement of invasion by viable rickettsiae for this host cell response.	Cytochalasin D	186-200	mitogen-activated protein kinase 14	Homo sapiens	76-79	
16979163-3	2006	We demonstrate that vinblastin and cytochalasin D, which respectively disorganize microtubules and actin microfilaments, trigger the activation of the p38/MAPKAP2 kinase pathway and lead to the specific alphaB-crystallin phosphorylation at serine 59.	Cytochalasin D	35-49	mitogen-activated protein kinase 14	Homo sapiens	151-154	
15137059-8	2004	Cytochalasin D also activated p38 MAP kinase, which pathway contributed to the cytochalasin D-induced increase in IL-8 production.	Cytochalasin D	0-14	mitogen-activated protein kinase 14	Homo sapiens	30-33	
15137059-8	2004	Cytochalasin D also activated p38 MAP kinase, which pathway contributed to the cytochalasin D-induced increase in IL-8 production.	Cytochalasin D	79-93	mitogen-activated protein kinase 14	Homo sapiens	30-33