PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6167026-0	1981	Angiotensin converting enzyme activity in mice after subacute bleomycin administration.	Bleomycin	62-71	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	0-29	
27928554-7	2016	Further, Bleo inducetion of both AGT protein and of caspase-9 were prevented by the ACE inhibitor lisinopril.	Bleomycin	9-13	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	84-87	
20651228-0	2010	Angiotensin-converting enzyme N-terminal inactivation alleviates bleomycin-induced lung injury.	Bleomycin	65-74	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	0-29	
20651228-9	2010	This study shows that the inactivation of the N-terminal catalytic site of ACE significantly reduced bleomycin-induced lung fibrosis and implicates AcSDKP in the mechanism of protection.	Bleomycin	101-110	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	75-78	
12003776-8	2002	These results suggest that ACE inhibitor treatment decreases lung injury and the development of pulmonary hypertension in bleomycin-treated mice.	Bleomycin	122-131	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	27-30	
12003776-1	2002	The present study was undertaken to investigate the effects of treatment with the angiotensin-converting enzyme (ACE) inhibitor enalapril in a mouse model of pulmonary hypertension induced by bleomycin.	Bleomycin	192-201	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	82-111	
12003776-1	2002	The present study was undertaken to investigate the effects of treatment with the angiotensin-converting enzyme (ACE) inhibitor enalapril in a mouse model of pulmonary hypertension induced by bleomycin.	Bleomycin	192-201	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	113-116