PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11438635-1 2001 UNLABELLED: Previous study of the bleomycin-induced lung injury model suggested that (111)In-labeled antirat intercellular adhesion molecule-1 (aICAM-1) might be a useful acute respiratory distress syndrome (ARDS) diagnostic agent. Bleomycin 34-43 intercellular adhesion molecule 1 Rattus norvegicus 109-142 15181831-8 2004 After intratracheal instillation of bleomycin, the number of positive staining cells for ICAM-1 expression significantly increased on day 1, reached the peak value on day 7, gradually decreased from then on, but still it was higher than that of the NS group on day 28 (P < 0.05). Bleomycin 36-45 intercellular adhesion molecule 1 Rattus norvegicus 89-95 9544689-0 1998 Early detection of bleomycin-induced lung injury in rat using indium-111-labeled antibody directed against intercellular adhesion molecule-1. Bleomycin 19-28 intercellular adhesion molecule 1 Rattus norvegicus 107-140 9544689-1 1998 UNLABELLED: We have investigated whether an (111)In-labeled mouse monoclonal antibody to rat intercellular adhesion molecule-1 ((111)In*aICAM-1) could detect lung injury early in rats treated with bleomycin. Bleomycin 197-206 intercellular adhesion molecule 1 Rattus norvegicus 93-126 9544689-10 1998 Lung ICAM-1 immunofluorescence intensity increased in the bleomycin-treated samples compared to uninjured lungs. Bleomycin 58-67 intercellular adhesion molecule 1 Rattus norvegicus 5-11 8574890-8 1995 Immunoelectron microscopy with preembedded rat lungs (bleomycin-exposed cases) confirmed the altered ICAM-1 distribution at the alveolar epithelial surface. Bleomycin 54-63 intercellular adhesion molecule 1 Rattus norvegicus 101-107 10806175-0 2000 Roles of ICAM-1 for abnormal leukocyte recruitment in the microcirculation of bleomycin-induced fibrotic lung injury. Bleomycin 78-87 intercellular adhesion molecule 1 Rattus norvegicus 9-15 19254480-1 2009 Previous study showed that aerosolized signal transducer and activator of transcription-1 (STAT1) antisense oligodeoxynucleotide (ASON) inhibited the expression of STAT1 and ICAM-1 mRNA and protein in alveolar macrophages (AMs) and decreased the concentrations of TGF-beta, PDGF and TNF-alpha in bronchioalveolar lavage fluid (BALF) in bleomycin (BLM)-induced rat pulmonary fibrosis. Bleomycin 336-345 intercellular adhesion molecule 1 Rattus norvegicus 174-180