PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34600594-11	2021	In the lung tissues of 1 mg/kg BLM-induced mice, RAGE expression was gradually decreased in 1- and 2 weeks in immunohistochemistry and western blot analysis, and 3 mg/kg of BLM-induced mice exhibited decreased RAGE levels while alpha-SMA expression was increased.	Bleomycin	31-34	advanced glycosylation end product-specific receptor	Mus musculus	49-53	
26545872-4	2015	The present in vivo study with RAGE null mice, we further confirmed the evidence that lack of RAGE evolves worse bleomycin-induced pulmonary fibrosis compared with control mice.	Bleomycin	113-122	advanced glycosylation end product-specific receptor	Mus musculus	94-98	
21487520-6	2011	found that RAGE null mice were protected from bleomycin-induced fibrosis and suggested the effect was due to a lack of HMGB1 induced RAGE signaling.	Bleomycin	46-55	advanced glycosylation end product-specific receptor	Mus musculus	11-15	
21487520-11	2011	A lack of RAGE was found to be protective against bleomycin injury in both in vivo and in vitro studies.	Bleomycin	50-59	advanced glycosylation end product-specific receptor	Mus musculus	10-14	
20333255-1	2010	BACKGROUND: The role of the receptor for advanced glycation end-products (RAGE) has been shown to differ in two different mouse models of asbestos and bleomycin induced pulmonary fibrosis.	Bleomycin	151-160	advanced glycosylation end product-specific receptor	Mus musculus	74-78	
17951314-3	2007	The aim of this study was to examine the contribution of RAGE during the acute inflammation and chronic fibrotic phases of lung injury induced by intratracheal instillation of bleomycin in mice.	Bleomycin	176-185	advanced glycosylation end product-specific receptor	Mus musculus	57-61	
17951314-4	2007	Bleomycin-induced lung fibrosis was evaluated in wild-type and RAGE-deficient (RAGE-/-) mice.	Bleomycin	0-9	advanced glycosylation end product-specific receptor	Mus musculus	79-87	
17951314-8	2007	In addition, bleomycin administration induced high mobility group box 1 (HMGB-1) production, one of the ligands of RAGE, from inflammatory cells that accumulated within the air space.	Bleomycin	13-22	advanced glycosylation end product-specific receptor	Mus musculus	115-119	
17951314-12	2007	Our results suggested that RAGE contributes to bleomycin-induced lung fibrosis through EMT and profibrotic cytokine production.	Bleomycin	47-56	advanced glycosylation end product-specific receptor	Mus musculus	27-31	
34600594-11	2021	In the lung tissues of 1 mg/kg BLM-induced mice, RAGE expression was gradually decreased in 1- and 2 weeks in immunohistochemistry and western blot analysis, and 3 mg/kg of BLM-induced mice exhibited decreased RAGE levels while alpha-SMA expression was increased.	Bleomycin	31-34	advanced glycosylation end product-specific receptor	Mus musculus	210-214	
34600594-11	2021	In the lung tissues of 1 mg/kg BLM-induced mice, RAGE expression was gradually decreased in 1- and 2 weeks in immunohistochemistry and western blot analysis, and 3 mg/kg of BLM-induced mice exhibited decreased RAGE levels while alpha-SMA expression was increased.	Bleomycin	173-176	advanced glycosylation end product-specific receptor	Mus musculus	49-53	
34600594-11	2021	In the lung tissues of 1 mg/kg BLM-induced mice, RAGE expression was gradually decreased in 1- and 2 weeks in immunohistochemistry and western blot analysis, and 3 mg/kg of BLM-induced mice exhibited decreased RAGE levels while alpha-SMA expression was increased.	Bleomycin	173-176	advanced glycosylation end product-specific receptor	Mus musculus	210-214