PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 3092662-5 1986 However, electrical or reflex stimulation of the renal nerves, intrarenal infusion of angiotensin II, or infusion of arginine vasopressin stimulates the release of vasodilator prostaglandins from the kidneys. Prostaglandins 176-190 arginine vasopressin Homo sapiens 126-137 3529950-6 1986 Vasoconstrictors such as angiotensin II, norepinephrine, and vasopressin increase production of the vasodilator prostaglandins, thereby preventing an overshoot of their action. Prostaglandins 112-126 arginine vasopressin Homo sapiens 61-72 3020336-5 1986 PG have been reported to inhibit ADH-stimulated water flow by inhibiting adenylcyclase. Prostaglandins 0-2 arginine vasopressin Homo sapiens 33-36 3020336-8 1986 PG and the AA cascade have been implicated as cellular modulators of the ADH hydroosmotic response. Prostaglandins 0-2 arginine vasopressin Homo sapiens 73-76 3080876-4 1986 The potential role for endogenous prostaglandins to antagonize the effect of antidiuretic hormone and to alter renal water excretion is also described. Prostaglandins 34-48 arginine vasopressin Homo sapiens 77-97 3006514-10 1986 That prostaglandin inhibitors also block the effect of PTH on vasopressin-stimulated water flow suggests that prostaglandin synthesis is required for the effect. Prostaglandins 5-18 arginine vasopressin Homo sapiens 62-73 3006514-10 1986 That prostaglandin inhibitors also block the effect of PTH on vasopressin-stimulated water flow suggests that prostaglandin synthesis is required for the effect. Prostaglandins 110-123 arginine vasopressin Homo sapiens 62-73 3006514-12 1986 Prostaglandin release, in turn, appears to mediate the inhibitory effect of PTH on vasopressin-stimulated water transport. Prostaglandins 0-13 arginine vasopressin Homo sapiens 83-94 3012727-5 1986 These studies suggest that the defect in renal concentrating ability seen as part of aminoglycoside nephrotoxicity could be due, in part, to an acidification-induced, prostaglandin-mediated resistance to the action of ADH. Prostaglandins 167-180 arginine vasopressin Homo sapiens 218-221 3031349-0 1986 Role of inhibitory and stimulative effects of prostaglandins on vasopressin-stimulated osmotic water flow in the toad bladder. Prostaglandins 46-60 arginine vasopressin Homo sapiens 64-75 2433528-2 1986 The formation of cyclic AMP (cAMP) by the adenylate cyclase system--consisting of the catalyst itself, the Ns and Ni proteins, and various hormone receptors--is stimulated by prostaglandins and adenosine, and is inhibited by alpha 2-adrenergic agonists, ADP, vasopressin, platelet-activating factor, and thrombin. Prostaglandins 175-189 arginine vasopressin Homo sapiens 259-270 3472330-8 1986 The responses of prostaglandin levels in perilymph to these experimental conditions (aspirin injection, antidiuretic hormone or epinephrine infusion) suggest that prostaglandins may play an important role under physiological conditions. Prostaglandins 17-30 arginine vasopressin Homo sapiens 104-124 3487106-4 1986 In the kidney, prostaglandins PGE2 and PGI2 modulate the vasoconstrictor effects of angiotensin II, norepinephrine, and vasopressin. Prostaglandins 15-29 arginine vasopressin Homo sapiens 120-131 3472330-8 1986 The responses of prostaglandin levels in perilymph to these experimental conditions (aspirin injection, antidiuretic hormone or epinephrine infusion) suggest that prostaglandins may play an important role under physiological conditions. Prostaglandins 163-177 arginine vasopressin Homo sapiens 104-124 2579584-1 1985 Prostaglandin (PG) inhibits the hydroosmotic effect of vasopressin. Prostaglandins 0-13 arginine vasopressin Homo sapiens 55-66 3922218-3 1985 Inhibition of PG synthesis by nonsteroidal antiinflammatory drugs (NSAID) augments the vasoconstrictor response to exogenous pressors such as angiotensin II, arginine vasopressin (AVP), and fludrocortisone. Prostaglandins 14-16 arginine vasopressin Homo sapiens 167-178 3856399-1 1985 Vasopressin stimulates renal prostaglandin (PGE2) production at several loci and in turn PGE2 modulates the antidiuresis. Prostaglandins 29-42 arginine vasopressin Homo sapiens 0-11 3846709-7 1985 The addition of prostaglandin E2 caused vasopressin-stimulated water flow to decrease (17.5 +/- 2.7 to 5.71 +/- 1.0 microliter/min) while the inhibition of endogenous prostaglandins caused vasopressin-stimulated water flow to increase (26.7 +/- 3.4 to 39.2 +/- 3.5 microliters/min). Prostaglandins 167-181 arginine vasopressin Homo sapiens 40-51 2579584-1 1985 Prostaglandin (PG) inhibits the hydroosmotic effect of vasopressin. Prostaglandins 15-17 arginine vasopressin Homo sapiens 55-66 3846709-8 1985 Thus, the inhibitory effects of prostaglandins and kinins on vasopressin-stimulated water flow are independent of one another. Prostaglandins 32-46 arginine vasopressin Homo sapiens 61-72 3846709-0 1985 Independent action of prostaglandins and kinins on vasopressin-stimulated water flow. Prostaglandins 22-36 arginine vasopressin Homo sapiens 51-62 3846709-1 1985 The kallikrein-kinin and the prostaglandin systems are both important modifiers of vasopressin action. Prostaglandins 29-42 arginine vasopressin Homo sapiens 83-94 2579584-12 1985 This may initiate PG synthesis and provide a link among VP, cAMP, and calcium. Prostaglandins 18-20 arginine vasopressin Homo sapiens 56-58 2579584-13 1985 A double-reciprocal feedback is proposed, whereby VP stimulates PG synthesis in a cAMP-independent manner and also inhibits PG synthesis in a cAMP-dependent manner. Prostaglandins 64-66 arginine vasopressin Homo sapiens 50-52 2579584-13 1985 A double-reciprocal feedback is proposed, whereby VP stimulates PG synthesis in a cAMP-independent manner and also inhibits PG synthesis in a cAMP-dependent manner. Prostaglandins 124-126 arginine vasopressin Homo sapiens 50-52 3918461-5 1985 The addition of a very low dose of antidiuretic hormone (ADH) (0.125 mU/ml) during prostaglandin synthesis inhibition increased luminal membrane water permeability to the same level as maximal stimulation with ADH, while tissue water permeability failed to increase proportionately. Prostaglandins 83-96 arginine vasopressin Homo sapiens 35-55 3918461-5 1985 The addition of a very low dose of antidiuretic hormone (ADH) (0.125 mU/ml) during prostaglandin synthesis inhibition increased luminal membrane water permeability to the same level as maximal stimulation with ADH, while tissue water permeability failed to increase proportionately. Prostaglandins 83-96 arginine vasopressin Homo sapiens 57-60 3011158-7 1985 In order to clarify whether calcium ions act directly on the adenylate cyclase system or activate a more complex pathway, we studied the interaction between calcium and prostaglandins in modulating the hydrosmotic response to vasopressin. Prostaglandins 169-183 arginine vasopressin Homo sapiens 226-237 2939907-5 1985 Using this technique, I found that the effect of the prostaglandin inhibitor Naproxen to increase vasopressin-stimulated water flow is due to increased luminal membrane permeability. Prostaglandins 53-66 arginine vasopressin Homo sapiens 98-109 3011158-9 1985 Furthermore, the inhibition of prostaglandin biosynthesis by hydrocortisone (a phospholipase inhibitor) or by indomethacin and naproxen (agents that inhibit arachidonic acid oxygenase) results in augmented vasopressin-stimulated water flow and prevents the inhibitory effect of the ionophore. Prostaglandins 31-44 arginine vasopressin Homo sapiens 206-217 2412098-0 1985 Modulation of vasopressin-induced water permeability of the cortical collecting tubule by endogenous and exogenous prostaglandins. Prostaglandins 115-129 arginine vasopressin Homo sapiens 14-25 2412098-1 1985 These experiments were designed to test the thesis that prostaglandins produced by the cortical collecting tubule cells could modulate the vasopressin-induced osmotic water permeability (Pf). Prostaglandins 56-70 arginine vasopressin Homo sapiens 139-150 2997891-3 1985 This inhibitory effect of TFP on the ADH-stimulated osmotic water flow persisted in the presence of naproxen (10(-5) M), a known inhibitor of prostaglandin synthesis. Prostaglandins 142-155 arginine vasopressin Homo sapiens 37-40 6426373-1 1984 Vasodilatory renal prostaglandins, especially PGE2 and PGI2, maintain renal blood flow and glomerular filtration rate under certain circumstances, especially clinical and experimental conditions accompanied by renal vasoconstriction and increased plasma concentrations of catecholamines, angiotensin, and vasopressin. Prostaglandins 19-33 arginine vasopressin Homo sapiens 305-316 6587004-3 1984 One such mechanism may be an impairment in prostaglandin (PGE2) excretion resulting in unopposed activity of antidiuretic hormone (ADH). Prostaglandins 43-56 arginine vasopressin Homo sapiens 109-129 6587004-3 1984 One such mechanism may be an impairment in prostaglandin (PGE2) excretion resulting in unopposed activity of antidiuretic hormone (ADH). Prostaglandins 43-56 arginine vasopressin Homo sapiens 131-134 6573182-0 1983 Involvement of prostaglandins in vasopressin stimulation of the human uterus. Prostaglandins 15-29 arginine vasopressin Homo sapiens 33-44 6091412-5 1984 Indomethacin inhibition of PG-synthesis was performed, and after high-Na provocation, an increase of vasopressin and cyclic AMP excretion by 15% and 376% more than that without indomethacin was found. Prostaglandins 27-29 arginine vasopressin Homo sapiens 101-112 6091412-7 1984 The results confirmed the assumption that renal prostaglandins are modulators of renin release from the kidney and that they alternate vasopressin effect on urine concentration, most probably through adenylate cyclase-cAMP system. Prostaglandins 48-62 arginine vasopressin Homo sapiens 135-146 6741563-8 1984 A negative correlation between renal PG production and VP plasma levels and excretion was demonstrated during the changes of dietary sodium intake. Prostaglandins 37-39 arginine vasopressin Homo sapiens 55-57 6573182-1 1983 The involvement of prostaglandins (PG) in the vasopressin (VP) action on the human uterus was investigated in healthy women during three menstruations. Prostaglandins 19-33 arginine vasopressin Homo sapiens 46-57 6573182-1 1983 The involvement of prostaglandins (PG) in the vasopressin (VP) action on the human uterus was investigated in healthy women during three menstruations. Prostaglandins 35-37 arginine vasopressin Homo sapiens 46-57 6573182-1 1983 The involvement of prostaglandins (PG) in the vasopressin (VP) action on the human uterus was investigated in healthy women during three menstruations. Prostaglandins 35-37 arginine vasopressin Homo sapiens 59-61 6281092-7 1982 The role of cAMP phosphodiesterase, hyaluronidase, aldosterone, prostaglandins and other physiologically active substances in the action of ADH has been discussed. Prostaglandins 64-78 arginine vasopressin Homo sapiens 140-143 6816073-0 1982 Vasopressin stimulates prostaglandin and thromboxane synthesis in toad bladder epithelial cells. Prostaglandins 23-36 arginine vasopressin Homo sapiens 0-11 6801089-6 1982 Preincubation with prostaglandin synthesis inhibitors reversed in major part the effect of serosal acidification on the response to submaximal concentrations of vasopressin and completely abolished the effect of pH on near maximal concentrations of the hormone. Prostaglandins 19-32 arginine vasopressin Homo sapiens 161-172 6801089-8 1982 These studies establish that increased basal PGE2 synthesis plays a critical role in the pH dependence of the hydroosmotic response to vasopressin and demonstrate that factors that modulate the response to vasopressin may exert this effect by changing the basal rate of prostaglandin synthesis. Prostaglandins 270-283 arginine vasopressin Homo sapiens 135-146 6801089-8 1982 These studies establish that increased basal PGE2 synthesis plays a critical role in the pH dependence of the hydroosmotic response to vasopressin and demonstrate that factors that modulate the response to vasopressin may exert this effect by changing the basal rate of prostaglandin synthesis. Prostaglandins 270-283 arginine vasopressin Homo sapiens 206-217 6300191-1 1983 The effect of 8-L-arginine vasopressin (AVP) on biosynthesis of prostaglandins in human mononuclear phagocytes was examined. Prostaglandins 64-78 arginine vasopressin Homo sapiens 14-38 6300191-1 1983 The effect of 8-L-arginine vasopressin (AVP) on biosynthesis of prostaglandins in human mononuclear phagocytes was examined. Prostaglandins 64-78 arginine vasopressin Homo sapiens 40-43 6300191-2 1983 AVP, oxytocin, and deamino-(8-D-arginine) vasopressin (dDAVP) affected prostaglandin biosynthesis in a rank order that parallels their pressor but not antidiuretic activity (AVP greater than oxytocin greater than dDAVP). Prostaglandins 71-84 arginine vasopressin Homo sapiens 0-3 6300191-2 1983 AVP, oxytocin, and deamino-(8-D-arginine) vasopressin (dDAVP) affected prostaglandin biosynthesis in a rank order that parallels their pressor but not antidiuretic activity (AVP greater than oxytocin greater than dDAVP). Prostaglandins 71-84 arginine vasopressin Homo sapiens 57-60 6301507-0 1983 Role of calcium and prostaglandins in the antidiuretic hormone response. Prostaglandins 20-34 arginine vasopressin Homo sapiens 42-62 6301507-6 1983 Indomethacin, a prostaglandin synthesis inhibitor, prevented the inhibitory actions of the IP on the ADH-mediated response. Prostaglandins 16-29 arginine vasopressin Homo sapiens 101-104 6301507-7 1983 Collectively, these observations suggest a key role for cellular calcium in modulating the actions of antidiuretic hormone and are consistent with the hypothesis that the ionophore, through increasing intracellular calcium, stimulates the synthesis of prostaglandins which have a negative feedback on adenylcyclase. Prostaglandins 252-266 arginine vasopressin Homo sapiens 102-122 6294273-10 1983 These studies suggest that prostaglandins and other agents which alter the effect of vasopressin in the isolated toad bladder may elicit their effects in part by influencing the calcium concentration at some critical site. Prostaglandins 27-41 arginine vasopressin Homo sapiens 85-96 6777397-1 1980 Prostaglandins are important modulators of the action of vasopressin. Prostaglandins 0-14 arginine vasopressin Homo sapiens 57-68 6290865-0 1982 Inhibition of prostaglandin synthesis antagonizes the colchicine-induced reduction of vasopressin-stimulated water flow in the toad urinary bladder. Prostaglandins 14-27 arginine vasopressin Homo sapiens 86-97 7323209-0 1981 The effect of vasopressin in water-loaded hypokalemic patients is prostaglandin-independent. Prostaglandins 66-79 arginine vasopressin Homo sapiens 14-25 7323209-4 1981 To evaluate the role of prostaglandins on the renal action of vasopressin in hypokalemia, six subjects with hypokalemia of various etiologies were studied in a control, drug-free condition and again after 3 to 6 days of indomethacin (100 mg/day). Prostaglandins 24-38 arginine vasopressin Homo sapiens 62-73 6265142-14 1981 In patients with central diabetes insipidus the urinary concentrating response to desamino-[Arg8]vasopressin is enhanced during inhibition of prostaglandin synthesis without changes in urinary excretion of cyclic AMP. Prostaglandins 142-155 arginine vasopressin Homo sapiens 97-108 7018256-8 1981 The medullary prostaglandins thus antagonize the ultimate action of vasopressin. Prostaglandins 14-28 arginine vasopressin Homo sapiens 68-79 6275715-2 1981 Both water flow and activation of cAMP-kinase in response to vasopressin were enhanced after prostaglandin inhibition, consistent with inhibition of vasopressin-induced cAMP generation by endogenous prostaglandins. Prostaglandins 93-106 arginine vasopressin Homo sapiens 61-72 6275715-2 1981 Both water flow and activation of cAMP-kinase in response to vasopressin were enhanced after prostaglandin inhibition, consistent with inhibition of vasopressin-induced cAMP generation by endogenous prostaglandins. Prostaglandins 93-106 arginine vasopressin Homo sapiens 149-160 6275715-2 1981 Both water flow and activation of cAMP-kinase in response to vasopressin were enhanced after prostaglandin inhibition, consistent with inhibition of vasopressin-induced cAMP generation by endogenous prostaglandins. Prostaglandins 199-213 arginine vasopressin Homo sapiens 61-72 6275715-2 1981 Both water flow and activation of cAMP-kinase in response to vasopressin were enhanced after prostaglandin inhibition, consistent with inhibition of vasopressin-induced cAMP generation by endogenous prostaglandins. Prostaglandins 199-213 arginine vasopressin Homo sapiens 149-160 7254732-0 1981 Effects of prostaglandin inhibition on vasopressin levels in women with primary dysmenorrhea. Prostaglandins 11-24 arginine vasopressin Homo sapiens 39-50 7254732-2 1981 In the treated group the AVP concentration was about 4 times higher than that in the controls, and the AVP level remained unchanged after almost complete pain relief had been obtained by treatment with the prostaglandin synthesis inhibitor. Prostaglandins 206-219 arginine vasopressin Homo sapiens 25-28 7254732-4 1981 These results indicate that increased secretion of AVP may play an etiologic role in dysmenorrhea, activating the uterus via a final step of prostaglandin synthesis an release. Prostaglandins 141-154 arginine vasopressin Homo sapiens 51-54 6267350-0 1981 Vasopressin-prostaglandin interactions in the regulation of epithelial cell permeability to water. Prostaglandins 12-25 arginine vasopressin Homo sapiens 0-11 6777397-2 1980 Others researchers have proposed that vasopressin stimulates prostaglandin synthesis, completing a negative feedback loop and thereby limiting vasopressin"s antidiuretic effect. Prostaglandins 61-74 arginine vasopressin Homo sapiens 38-49 6777397-2 1980 Others researchers have proposed that vasopressin stimulates prostaglandin synthesis, completing a negative feedback loop and thereby limiting vasopressin"s antidiuretic effect. Prostaglandins 61-74 arginine vasopressin Homo sapiens 143-154 6777397-12 1980 Furthermore, because AVP appears to increase prostaglandin synthesis by the intact kidney, we suggest that cells other than those of the collecting tubule are responsible for the increased prostaglandin production. Prostaglandins 45-58 arginine vasopressin Homo sapiens 21-24 7362800-6 1980 An interaction of PG and other endogenous myometrial stimulants such as VP might be important in the aetiology of spontaneous abortion and the combination of PG with VP-like substances might be of value in therapeutic abortion during the first trimester. Prostaglandins 18-20 arginine vasopressin Homo sapiens 72-74 4356205-0 1973 Release of prostaglandins from isolated cat spleen by angiotensin and vasopressin. Prostaglandins 11-25 arginine vasopressin Homo sapiens 70-81 7000470-2 1980 From all those results the picture of a rather tight interplay between renal prostaglandins, renin-angiotensin, aldosterone, and antidiuretic hormone (ADH) emerges, whereby renal prostaglandins synthesis appears to be partially regulated by angiotensin II and ADH; renal prostaglandins antagonize the biological effects of these peptide hormones, and directly or indirectly affect in their turn the release of renin and ADH. Prostaglandins 179-193 arginine vasopressin Homo sapiens 151-154 7000470-2 1980 From all those results the picture of a rather tight interplay between renal prostaglandins, renin-angiotensin, aldosterone, and antidiuretic hormone (ADH) emerges, whereby renal prostaglandins synthesis appears to be partially regulated by angiotensin II and ADH; renal prostaglandins antagonize the biological effects of these peptide hormones, and directly or indirectly affect in their turn the release of renin and ADH. Prostaglandins 179-193 arginine vasopressin Homo sapiens 151-154 380250-4 1979 Other mechanisms of possible importance such as ovarian hormones, cervical factors, vasopressin, nerves, and psychological factors can well act ultimately through prostaglandin release but an action directly on the myometrium and blood flow may also occur. Prostaglandins 163-176 arginine vasopressin Homo sapiens 84-95 744021-5 1978 This suggests that vasopressin may be acting by stimulating prostaglandin biosynthesis. Prostaglandins 60-73 arginine vasopressin Homo sapiens 19-30 744021-6 1978 Cortisol and lithium may exert their inhibitory effects by preventing the activation of prostaglandin synthesis by vasopressin. Prostaglandins 88-101 arginine vasopressin Homo sapiens 115-126 212251-1 1978 Current experimental evidence indicates that endogenous renal medullary prostaglandins modulate the antidiuretic response to vasopressin in the mammalian kidney. Prostaglandins 72-86 arginine vasopressin Homo sapiens 125-136 212251-2 1978 The predominant effect of prostaglandins is to attenuate the antidiuretic response to vasopressin; inhibition of prostaglandin synthesis potentiates the renal effect of vasopressin. Prostaglandins 26-40 arginine vasopressin Homo sapiens 86-97 631967-5 1978 The results can be interpreted on the basis of either a direct "vasopressin-like" action of indomethacin or abolishment of the peripheral vasopressin--prostaglandin interaction. Prostaglandins 151-164 arginine vasopressin Homo sapiens 138-149 197384-3 1977 We have also found that various hormones, including prolactin, vasopressin and angiotensin when present in physiological concentrations can be potent stimulators of prostaglandin synthesis. Prostaglandins 165-178 arginine vasopressin Homo sapiens 63-74 456612-4 1979 Centrally administered angiotensin II can stimulate the release of vasopressin, a response that may be partially mediated by brain prostaglandins. Prostaglandins 131-145 arginine vasopressin Homo sapiens 67-78 214803-6 1978 Since this was not accompanied by respective changes in urinary excretion of cyclic adenosine monophosphate (cAMP) mechanisms other than PG-antagonism of vasopressin, such as decreased medullary washout of solute, may contribute to enhanced renal concentrating ability following inhibition of PG-synthesis with indomethacin. Prostaglandins 137-139 arginine vasopressin Homo sapiens 154-165 212251-2 1978 The predominant effect of prostaglandins is to attenuate the antidiuretic response to vasopressin; inhibition of prostaglandin synthesis potentiates the renal effect of vasopressin. Prostaglandins 26-39 arginine vasopressin Homo sapiens 86-97 212251-3 1978 Prostaglandins likely antagonize the renal effects of vasopressin at the cellular level of hormone-dependent cyclic adenosine 3,5-monophosphate metabolism, but the exact molecular mechanism is not known. Prostaglandins 0-14 arginine vasopressin Homo sapiens 54-65 212251-5 1978 Vasopressin itself could stimulate intrarenal prostaglandin synthesis; this effect may represent a negative-feedback regulatory pathway for the antidiuretic response to the hormone. Prostaglandins 46-59 arginine vasopressin Homo sapiens 0-11 1150880-6 1975 In another group of studies the second dose of vasopressin was preceded by the administration of a potent inhibitor of prostaglandin synthesis, indomethacin (2 mg/kg). Prostaglandins 119-132 arginine vasopressin Homo sapiens 47-58 1150880-11 1975 These results therefore implicate a physiological role of prostaglandin in modulating the hydroosmotic effect of vasopressin in the mammalian kidney. Prostaglandins 58-71 arginine vasopressin Homo sapiens 113-124 20606409-10 2010 Vasopressin has many effects on immune function such as altering cytokines, neuroimmunity, prostaglandins, humoral immunity and immune cells. Prostaglandins 91-105 arginine vasopressin Homo sapiens 0-11 21143292-2 2011 In mature kidneys, prostaglandins counteract the effect of AVP on AQP2 expression at functional sites. Prostaglandins 19-33 arginine vasopressin Homo sapiens 59-62 27372869-4 2016 IL-1beta was found to stimulate the secretion of antidiuretic hormone (ADH) from the posterior pituitary gland either by action of prostaglandins or indirectly by causing the release of IL-6. Prostaglandins 131-145 arginine vasopressin Homo sapiens 49-69 27372869-4 2016 IL-1beta was found to stimulate the secretion of antidiuretic hormone (ADH) from the posterior pituitary gland either by action of prostaglandins or indirectly by causing the release of IL-6. Prostaglandins 131-145 arginine vasopressin Homo sapiens 71-74 20606409-11 2010 For example, vasopressin decreases sepsis-induced pulmonary inflammation, could have renal anti-inflammatory effects and may decrease prostaglandin levels in a dose-dependent manner. Prostaglandins 134-147 arginine vasopressin Homo sapiens 13-24 18090373-12 2008 CONCLUSION: The results provide functional evidence that aspirin at high concentrations and the COX-2 selective inhibitor nimesulide potentiate the contractile response of gastroepiploic artery to vasopressin, thus suggesting the release of relaxant prostaglandins by the peptide. Prostaglandins 250-264 arginine vasopressin Homo sapiens 197-208 18310512-1 2008 Historically, the vasodilatory prostanoids, especially prostacyclin and prostaglandin E(2), are believed to contribute significantly to the regulation of normal vascular tone and blood pressure (BP), primarily by counteracting the prevailing effects of the systemic vasoconstrictor systems, including angiotensin II, the catecholamines, and vasopressin. Prostaglandins 31-42 arginine vasopressin Homo sapiens 341-352 12614377-9 2003 By inhibiting prostaglandin synthesis, non-steroid anti-inflammatory agents (NSAIDs) potentiate the effect of water reabsorption in the renal tubules of vasopressin, therefore enhancing water retention. Prostaglandins 14-27 arginine vasopressin Homo sapiens 153-164 15613736-1 2004 The review presents our results on the regulatory role of prostaglandins (PG) and nitric oxide (NO) in the activation of hypothalamic-pituitary-adrenal (HPA) axis by cholinergic, adrenergic and histaminergic systems and by neurohormones: corticotropin-releasing hormone (CRH) and arginine vasopressin (AVP) under basal conditions. Prostaglandins 58-72 arginine vasopressin Homo sapiens 289-300 15613736-1 2004 The review presents our results on the regulatory role of prostaglandins (PG) and nitric oxide (NO) in the activation of hypothalamic-pituitary-adrenal (HPA) axis by cholinergic, adrenergic and histaminergic systems and by neurohormones: corticotropin-releasing hormone (CRH) and arginine vasopressin (AVP) under basal conditions. Prostaglandins 74-76 arginine vasopressin Homo sapiens 289-300 15613736-18 2004 The vasopressin-stimulated increase in HPA response, was considerably reduced by the inhibition of PG synthesis by both COX antagonists while inhibition of NO synthesis by NOS blockers greatly enhanced this response. Prostaglandins 99-101 arginine vasopressin Homo sapiens 4-15 16641851-5 2006 Plasma levels of vasopressin appear to be higher in women with dysmenorrhea suggesting a possible aetiological role in the uterine prostaglandin synthesis. Prostaglandins 131-144 arginine vasopressin Homo sapiens 17-28 9353864-9 1997 These data clearly indicate that the increased renal synthesis of vasodilating PGs has a relevant role in maintaining renal hemodynamics, sodium and water excretion in a clinical setting characterized by a reduction of effective plasma volume and a striking activation of the major vasoconstricting systems, namely the renin-angiotensin-aldosterone, the sympathetic nervous system, and vasopressin. Prostaglandins 79-82 arginine vasopressin Homo sapiens 386-397 7804750-6 1994 In addition, angiotensin II-mediated and arginine vasopressin-mediated mesangial cell Ca2+ signals and contraction are attenuated via prostaglandin production by the mesangial cells themselves when the ambient Cl- concentration is reduced. Prostaglandins 134-147 arginine vasopressin Homo sapiens 50-61 8957201-7 1996 Vasopressin causes prostaglandin-mediated dilation of human renal arteries only if V1-receptor blockade is present. Prostaglandins 19-32 arginine vasopressin Homo sapiens 0-11 8201652-0 1994 A case of familial central diabetes insipidus: the response of urinary prostaglandins to 1-deamino-8-D-arginine vasopressin. Prostaglandins 71-85 arginine vasopressin Homo sapiens 112-123 8166228-3 1994 The vasoactive peptides, arginine vasopressin (AVP) or angiotensin II (ANG II), stimulated secretion of both immunoreactive ET-1 and prostanoids from HBEC by a receptor-mediated induction of phospholipase C (PLC) and PLA2. Prostaglandins 133-144 arginine vasopressin Homo sapiens 34-45 1647232-0 1991 [Stimulation by vasopressin of hydrolysis of phosphatidylinositol- 4,5-diphosphate and its relation to prostaglandin biosynthesis in the bladder epithelium of frogs]. Prostaglandins 103-116 arginine vasopressin Homo sapiens 16-27 8010758-3 1994 These stimulatory effects are damped by the action of several hormones, including vasopressin itself, which activate inhibitory G proteins, stimulate phospholipid breakdown, increase prostaglandin production, raise intracellular Ca2+, activate protein kinase C, stimulate tyrosine kinases, and raise cGMP. Prostaglandins 183-196 arginine vasopressin Homo sapiens 82-93 8198803-6 1993 The results show that: 1) AT II and cortical vasodilating prostanoids exerted opposite effects on the preglomerular arteriolar tone; 2) medullary prostanoids antagonized the vasopressin effects. Prostaglandins 58-69 arginine vasopressin Homo sapiens 174-185 8198803-6 1993 The results show that: 1) AT II and cortical vasodilating prostanoids exerted opposite effects on the preglomerular arteriolar tone; 2) medullary prostanoids antagonized the vasopressin effects. Prostaglandins 146-157 arginine vasopressin Homo sapiens 174-185 8198803-7 1993 Therefore, in potassium depletion the decreased synthesis of cortical and medullary prostanoids, in the face of the increased generation of AT II, contributed to reducing the glomerular filtration rate and facilitate the expression of vasopressin action. Prostaglandins 84-95 arginine vasopressin Homo sapiens 235-246 1326711-4 1992 Several hormones (vasopressin, oxytocin, luteinizing hormone, follicle-stimulating hormone, cortisol, estradiol, and testosterone) can indirectly affect prostaglandin formation leading to reduced bone formation. Prostaglandins 153-166 arginine vasopressin Homo sapiens 18-29 1963096-8 1990 In contrast, vasopressin, which appears to stimulate prostaglandin production in amnion cells by a phospholipase C-dependent mechanism, was equipotent in stimulating PGE2 production by amnion cells on Day 2 and Day 5 of culture, even though phospholipase C activity had declined by more than 75%. Prostaglandins 53-66 arginine vasopressin Homo sapiens 13-24 1699229-10 1990 Desensitization to vasopressin was observed in the presence and absence of indomethacin, indicating that it is independent of prostaglandin synthesis. Prostaglandins 126-139 arginine vasopressin Homo sapiens 19-30 2514664-4 1989 In the kidney, major stimuli of prostaglandin synthesis include vasoconstrictor hormones such as angiotensin II, vasopressin, endothelin and norepinephrine. Prostaglandins 32-45 arginine vasopressin Homo sapiens 113-124 2547832-11 1989 The direct vasodilatation induced by intraarterial AVP at higher doses is not mediated by prostaglandins but may involve cGMP-related mechanisms. Prostaglandins 90-104 arginine vasopressin Homo sapiens 51-54 3177653-7 1988 We propose that histidine increases the hydrosmotic response to vasopressin through at least two distinct mechanisms: 1) it decreases prostaglandin synthesis and thus increases luminal permeability; 2) it decreases the resistance to water movement of the tissues beneath the luminal membrane. Prostaglandins 134-147 arginine vasopressin Homo sapiens 64-75 3177653-0 1988 Effects of histidine on vasopressin action: role of decreased prostaglandin production. Prostaglandins 62-75 arginine vasopressin Homo sapiens 24-35