PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19442044-7	2009	Eicosanoid-independent pro-apoptotic pathways include enhanced lipid peroxidation, modulation of mitochondrial calcium homeostasis and enhanced production of reactive oxygen species as well as activation of p53.	Eicosanoids	0-10	tumor protein p53	Homo sapiens	207-210	
11390388-1	2001	Electrophilic eicosanoids of the J series, with their distinctive cross-conjugated alpha,beta-unsaturated ketone, inactivate genetically wild type tumor suppressor p53 in a manner analogous to prostaglandins of the A series.	Eicosanoids	14-25	tumor protein p53	Homo sapiens	164-167	
10908664-1	2000	The electrophilic eicosanoids prostaglandins A(1) or A(2) impaired p53-dependent transcription of endogenous genes and exogenous p53-luciferase reporter plasmids in RKO and HCT 116 colon cancer cells.	Eicosanoids	18-29	tumor protein p53	Homo sapiens	67-70	
10908664-1	2000	The electrophilic eicosanoids prostaglandins A(1) or A(2) impaired p53-dependent transcription of endogenous genes and exogenous p53-luciferase reporter plasmids in RKO and HCT 116 colon cancer cells.	Eicosanoids	18-29	tumor protein p53	Homo sapiens	129-132	
10908664-5	2000	We conclude that electrophilic eicosanoids impair the role of wild-type p53 as a guardian of genomic integrity by a process distinct from somatic mutation or viral oncoprotein binding.	Eicosanoids	31-42	tumor protein p53	Homo sapiens	72-75