PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22750268-3	2012	In the present work, we focused on reviewing the current knowledge about the dysregulation of the proteins/enzymes involved in the key regulatory steps of glucose transport, glycolysis, TCA cycle and glutaminolysis by several oncogenes including c-Myc and hypoxia inducible factor-1 (HIF-1) and tumor suppressor, p53, in cancer cells.	Tricarboxylic Acids	186-189	tumor protein p53	Homo sapiens	313-316	
31534224-8	2019	Enforcing the accumulation of alphaKG in p53-deficient PDAC cells through the inhibition of oxoglutarate dehydrogenase-an enzyme of the tricarboxylic acid cycle-specifically results in increased 5hmC, tumour-cell differentiation and decreased tumour-cell fitness.	Tricarboxylic Acids	136-154	tumor protein p53	Homo sapiens	41-44	
30765875-6	2019	RESULTS: Using a proteomic approach, we have shown that Notch3 is strongly involved in brivanib resistance through a p53-dependent regulation of enzymes of the tricarboxylic acid (TCA), both in vitro and in vivo.	Tricarboxylic Acids	160-178	tumor protein p53	Homo sapiens	117-120	
30765875-6	2019	RESULTS: Using a proteomic approach, we have shown that Notch3 is strongly involved in brivanib resistance through a p53-dependent regulation of enzymes of the tricarboxylic acid (TCA), both in vitro and in vivo.	Tricarboxylic Acids	180-183	tumor protein p53	Homo sapiens	117-120	
23334421-5	2013	We show that p53 represses the expression of the tricarboxylic-acid-cycle-associated malic enzymes ME1 and ME2 in human and mouse cells.	Tricarboxylic Acids	49-67	tumor protein p53	Homo sapiens	13-16