PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27258785-5	2016	Our results show that RIPK1 and RIPK3 participate in H2O2-induced cardiomyocytes necrosis.	Hydrogen Peroxide	53-57	receptor interacting serine/threonine kinase 1	Homo sapiens	22-27	
30784599-4	2019	By contrast, the same H2O2 burst stimulates the RIPK1-dependent apoptosis pathway in the absence of PrxII by inducing the destruction of cIAP1 in caveolar membrane.	Hydrogen Peroxide	22-26	receptor interacting serine/threonine kinase 1	Homo sapiens	48-53	
29608987-0	2018	RIP1 and RIP3 contribute to shikonin-induced glycolysis suppression in glioma cells via increase of intracellular hydrogen peroxide.	Hydrogen Peroxide	114-131	receptor interacting serine/threonine kinase 1	Homo sapiens	0-4	
29608987-7	2018	Notably, inhibition of RIP1 or RIP3 prevented intracellular H2O2 accumulation, which was correlated with preventing shikonin-induced downregulation of x-CT and depletion of GSH and cysteine.	Hydrogen Peroxide	60-64	receptor interacting serine/threonine kinase 1	Homo sapiens	23-27	
29608987-9	2018	Taken together, we demonstrated in this study that RIP1 and RIP3 contributed to shikonin-induced glycolysis suppression via increasing intracellular H2O2.	Hydrogen Peroxide	149-153	receptor interacting serine/threonine kinase 1	Homo sapiens	51-55	
30556404-6	2019	Results: MAM directly targets receptor-interacting protein 1 (RIP1) kinase in A549 and H1299 cells, which is responsible for reactive oxygen species (ROS, mainly hydrogen peroxide) generation.	Hydrogen Peroxide	162-179	receptor interacting serine/threonine kinase 1	Homo sapiens	30-60	
30556404-6	2019	Results: MAM directly targets receptor-interacting protein 1 (RIP1) kinase in A549 and H1299 cells, which is responsible for reactive oxygen species (ROS, mainly hydrogen peroxide) generation.	Hydrogen Peroxide	162-179	receptor interacting serine/threonine kinase 1	Homo sapiens	62-66	
34102211-4	2021	Furthermore, the expression of receptor-interacting protein 1 (RIP1) was remarkably upregulated in melanocytes treated with hydrogen peroxide (H2O2).	Hydrogen Peroxide	124-141	receptor interacting serine/threonine kinase 1	Homo sapiens	31-61	
24425875-4	2014	Furthermore, we found that, in RIP1 knockdown cells, the expression of the hydrogen peroxide-reducing enzyme catalase was dramatically reduced, which was associated with increased miR-146a expression.	Hydrogen Peroxide	75-92	receptor interacting serine/threonine kinase 1	Homo sapiens	31-35	
34102211-4	2021	Furthermore, the expression of receptor-interacting protein 1 (RIP1) was remarkably upregulated in melanocytes treated with hydrogen peroxide (H2O2).	Hydrogen Peroxide	124-141	receptor interacting serine/threonine kinase 1	Homo sapiens	63-67	
34102211-4	2021	Furthermore, the expression of receptor-interacting protein 1 (RIP1) was remarkably upregulated in melanocytes treated with hydrogen peroxide (H2O2).	Hydrogen Peroxide	143-147	receptor interacting serine/threonine kinase 1	Homo sapiens	31-61	
34102211-4	2021	Furthermore, the expression of receptor-interacting protein 1 (RIP1) was remarkably upregulated in melanocytes treated with hydrogen peroxide (H2O2).	Hydrogen Peroxide	143-147	receptor interacting serine/threonine kinase 1	Homo sapiens	63-67	
34102211-5	2021	Then, RIP1 intervention suppression and MLKL deficiency could significantly enhanced resistance of melanocytes to H2O2-induced necroptosis.	Hydrogen Peroxide	114-118	receptor interacting serine/threonine kinase 1	Homo sapiens	6-10	
34165415-3	2022	MAM fights cancer progression by inducing apoptosis, necroptosis, and deregulating signaling pathways through H2O2-induced JNK/iNOS/NO and MAPK, ERK1/2 pathways, JNK activation, and the RIP1/RIP3 complex.	Hydrogen Peroxide	110-114	receptor interacting serine/threonine kinase 1	Homo sapiens	186-190