PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33744257-0	2021	Long non-coding RNA KCNQ1OT1 promotes hydrogen peroxide-induced lens epithelial cell apoptosis and oxidative stress by regulating miR-223-3p/BCL2L2 axis.	Hydrogen Peroxide	38-55	BCL2 like 2	Homo sapiens	141-147	
33744257-13	2021	Additionally, BCL2L2 was a direct target of miR-223-3p, and miR-223-3p weakened H2O2-induced SRA01/04 cell apoptosis and oxidative stress by targeting BCL2L2.	Hydrogen Peroxide	80-84	BCL2 like 2	Homo sapiens	14-20	
33744257-13	2021	Additionally, BCL2L2 was a direct target of miR-223-3p, and miR-223-3p weakened H2O2-induced SRA01/04 cell apoptosis and oxidative stress by targeting BCL2L2.	Hydrogen Peroxide	80-84	BCL2 like 2	Homo sapiens	151-157	
21063685-6	2011	In addition, real-time PCR and western blot studies showed that Bcl-2 and Bcl-w expressions were decreased, and Bax expression was increased with H2O2 treatment.	Hydrogen Peroxide	146-150	BCL2 like 2	Homo sapiens	74-79	
30355909-10	2018	MiR-195 overexpression or BCL2L2 knockdown attenuated the effects of SNHG1 overexpression on cell viability, cell apoptosis and protein levels of cleaved caspase-3, cleaved caspase-9 and Bax in H2O2-treated HCMs.	Hydrogen Peroxide	194-198	BCL2 like 2	Homo sapiens	26-32	
21063685-10	2011	L-NBP treatment at dose of 10 muM inhibited H2O2-induced down-regulation of Bcl-2, Bcl-w, and PKCalpha but also attenuated the overexpression of Bax.	Hydrogen Peroxide	44-48	BCL2 like 2	Homo sapiens	83-88