PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31223425-7	2019	Thus, the induction of Nrf2-mediated oxidative and endoplasmic reticulum stress response, PPAR-alpha/RXR activation as well as production of peroxisomes, and prevention of apoptosis already during the first hour after CAP treatment are important cell strategies to overcome oxidative stress and to protect and maintain cell integrity and especially microtubule dynamics.	cap	218-221	NFE2 like bZIP transcription factor 2	Homo sapiens	23-27	
34029575-5	2021	In addition, CAP treatment reduced the infiltration of CD11c+ dendritic cells, CD3+ T cells, and CD8+ T cells; inhibited the release of chemokine (C-X-C motif) ligand 10 and cytokine interferon-gamma; and enhanced cellular resistance to oxidative stress and excessive immune response by enhancing the expression of the transcription factor Nrf2 and attenuating the activity of inducible nitric oxide synthase.	cap	13-16	NFE2 like bZIP transcription factor 2	Homo sapiens	340-344	
22521877-0	2012	Cap-independent Nrf2 translation is part of a lipoic acid-stimulated detoxification stress response.	cap	0-3	NFE2 like bZIP transcription factor 2	Homo sapiens	16-20