PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22395404-0	2012	Inhibitory effects of enalaprilat on rat cardiac fibroblast proliferation via ROS/P38MAPK/TGF-beta1 signaling pathway.	Enalaprilat	22-33	transforming growth factor, beta 1	Rattus norvegicus	90-99	
21795644-2	2011	Using cultured nephritic glomeruli from rats with anti-Thy-1-induced glomerulonephritis, the maximally effective dose of enalaprilate was determined to be 10(-4) M, which reduced mRNAs for transforming growth factor (TGF)-beta1, fibronectin (FN), and plasminogen activator inhibitor-1 (PAI-1) by 49, 65, and 56% and production of TGF-beta1 and FN proteins by 60 and 49%, respectively.	Enalaprilat	121-133	transforming growth factor, beta 1	Rattus norvegicus	189-227	
21795644-2	2011	Using cultured nephritic glomeruli from rats with anti-Thy-1-induced glomerulonephritis, the maximally effective dose of enalaprilate was determined to be 10(-4) M, which reduced mRNAs for transforming growth factor (TGF)-beta1, fibronectin (FN), and plasminogen activator inhibitor-1 (PAI-1) by 49, 65, and 56% and production of TGF-beta1 and FN proteins by 60 and 49%, respectively.	Enalaprilat	121-133	transforming growth factor, beta 1	Rattus norvegicus	330-339	
22395404-5	2012	It could be concluded that enalaprilat can inhibit the cardiac fibroblast proliferation induced by Ang II via blocking ROS/P38MAPK/TGF-beta(1) signaling pathways and the study provides a theoretical proof for the application of ACEIs in treating myocardial fibrosis and discovering the primary mechanism through which ACEIs inhibit CFb proliferation.	Enalaprilat	27-38	transforming growth factor, beta 1	Rattus norvegicus	131-142