PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18602458-4	2008	When ACh is applied at a low concentration (10 microM), the nAChR-mediated ion current is inhibited by a low concentration (10 microM) of ethylbenzene and p-xylene, but not by the same concentration of the non-ototoxic solvents.	ethylbenzene	138-150	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	60-65	
18602458-7	2008	These results demonstrate that low concentrations of the known ototoxic solvents ethylbenzene and p-xylene inhibit alpha9alpha10 nAChR-mediated ion currents, whereas the structurally related, non-ototoxic solvents m-xylene and o-xylene do not, indicating that the alpha9alpha10 nAChR is a potential target for solvent-induced ototoxicity.	ethylbenzene	81-93	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	129-134	
18602458-7	2008	These results demonstrate that low concentrations of the known ototoxic solvents ethylbenzene and p-xylene inhibit alpha9alpha10 nAChR-mediated ion currents, whereas the structurally related, non-ototoxic solvents m-xylene and o-xylene do not, indicating that the alpha9alpha10 nAChR is a potential target for solvent-induced ototoxicity.	ethylbenzene	81-93	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	278-283