PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 16182431-9 2005 Lung ACE activity decreased after paraquat administration and the decrement was maximal on day 7. Paraquat 34-42 angiotensin I converting enzyme Rattus norvegicus 5-8 20465954-0 2010 [Expression of angiotensin converting enzyme and angiotensin converting enzyme 2 gene in lung of paraquat poisoning rats and protection of sodium dimercaptopropane sulfonate]. Paraquat 97-105 angiotensin I converting enzyme Rattus norvegicus 15-44 20465954-10 2010 As compared with paraquat poisoned group, the expressions of ACE mRNA in lung tissue of rats in NA-DMPS protected group increased significantly at 24 h (0.739 +/- 0.558) and 3 d (0.749 +/- 0.414) (P < 0.05), while the expressions of ACE2 mRNA increased markedly on 3 d (0.584 +/- 0.345) and 7 d (0.493 +/- 0.292) (P < 0.05). Paraquat 17-25 angiotensin I converting enzyme Rattus norvegicus 61-64 17265423-3 2007 This study examined the effects of two angiotensin-converting enzyme inhibitors, captopril and enalapril, on paraquat-induced pulmonary fibrosis in rats, through biochemical and histopathological parameters. Paraquat 109-117 angiotensin I converting enzyme Rattus norvegicus 39-68 10666014-6 2000 In addition, the data also demonstrated that PQ caused a decrease in ACE and glutathione levels and an increase in levels of TBARS and myeloperoxidase (MPO) activity in the lung. Paraquat 45-47 angiotensin I converting enzyme Rattus norvegicus 69-72 10666014-4 2000 A single intraperitoneal injection of PQ (50 mg/kg) resulted in a significant rise in the levels of protein, angiotensin converting enzyme (ACE), alkaline phosphatase (AKP), N-acetyl-beta-D-glucosaminidase (NAG) and thiobarbituric acid reactive substances (TBARS), and neutrophils in the bronchoalveolar lavage fluid (BALF), while a decrease in glutathione levels. Paraquat 38-40 angiotensin I converting enzyme Rattus norvegicus 109-138 10666014-4 2000 A single intraperitoneal injection of PQ (50 mg/kg) resulted in a significant rise in the levels of protein, angiotensin converting enzyme (ACE), alkaline phosphatase (AKP), N-acetyl-beta-D-glucosaminidase (NAG) and thiobarbituric acid reactive substances (TBARS), and neutrophils in the bronchoalveolar lavage fluid (BALF), while a decrease in glutathione levels. Paraquat 38-40 angiotensin I converting enzyme Rattus norvegicus 140-143 10666014-9 2000 In addition, PQ induced reduction in lung ACE and BAL cell and lung glutathione levels was abolished by curcumin treatment. Paraquat 13-15 angiotensin I converting enzyme Rattus norvegicus 42-45 8937465-5 1996 Lungs of paraquat-challenged animals were damaged extensively as evidenced by increases in lung weight, indicative of edema, and decreases in lung activities of angiotensin converting enzyme (ACE) and alkaline phosphatase (AKP), indicative of endothelial and alveolar type II epithelial cell injuries, respectively. Paraquat 9-17 angiotensin I converting enzyme Rattus norvegicus 161-190 8937465-5 1996 Lungs of paraquat-challenged animals were damaged extensively as evidenced by increases in lung weight, indicative of edema, and decreases in lung activities of angiotensin converting enzyme (ACE) and alkaline phosphatase (AKP), indicative of endothelial and alveolar type II epithelial cell injuries, respectively. Paraquat 9-17 angiotensin I converting enzyme Rattus norvegicus 192-195 8937465-6 1996 While the pretreatment of rats with alpha-tocopherol liposomes or liposomes containing both alpha-tocopherol and GSH significantly attenuated paraquat-induced changes in lung ACE activity to more or less the same extent, the bifunctional liposomal preparation conferred additional protection to alveolar type II epithelial cells, as evidenced by a significantly higher pulmonary AKP activity. Paraquat 142-150 angiotensin I converting enzyme Rattus norvegicus 175-178 8719170-5 1995 Results of this study showed that lungs of animals treated with paraquat were extensively damaged, as evidenced by significant increases in lung weight and decreases in lung angiotensin converting enzyme (ACE) and alkaline phosphatase enzyme (AKP) activities. Paraquat 64-72 angiotensin I converting enzyme Rattus norvegicus 174-203 8719170-5 1995 Results of this study showed that lungs of animals treated with paraquat were extensively damaged, as evidenced by significant increases in lung weight and decreases in lung angiotensin converting enzyme (ACE) and alkaline phosphatase enzyme (AKP) activities. Paraquat 64-72 angiotensin I converting enzyme Rattus norvegicus 205-208 8719170-8 1995 On the other hand, pretreatment of rats with alpha-tocopherol liposomes, 24 h prior to paraquat challenge, attenuated paraquat-induced changes in ACE, AKP and myeloperoxidase activities but failed to prevent increases in lung weight. Paraquat 118-126 angiotensin I converting enzyme Rattus norvegicus 146-149 1333208-3 1992 Results of this study showed that lungs of animals treated with paraquat were damaged extensively as evidenced by an increase in lung weight and a significant reduction in lung angiotensin-converting enzyme (ACE) activity and cytochrome P450 concentration. Paraquat 64-72 angiotensin I converting enzyme Rattus norvegicus 177-206 1333208-3 1992 Results of this study showed that lungs of animals treated with paraquat were damaged extensively as evidenced by an increase in lung weight and a significant reduction in lung angiotensin-converting enzyme (ACE) activity and cytochrome P450 concentration. Paraquat 64-72 angiotensin I converting enzyme Rattus norvegicus 208-211 1333208-6 1992 On the other hand, pretreatment of rats with DPPC/alpha-tocopherol liposomes 24 hr prior to paraquat challenge resulted in a significant increase in pulmonary alpha-tocopherol concentrations and antagonized paraquat-induced changes in lipid peroxidation, GSH/GSSG ratio, lung ACE activity and cytochrome P450 concentrations. Paraquat 207-215 angiotensin I converting enzyme Rattus norvegicus 276-279