PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18056701-6	2008	Evaluation of Bcl-2 family members showed that PQ induced high levels of Bak, Bid, BNip3, and Noxa.	Paraquat	47-49	B cell leukemia/lymphoma 2	Mus musculus	14-19	
20937379-7	2010	Paraquat promoted overt apoptosis and ER stress as evidenced by increased caspase-3 activity, apoptosis, and ER stress markers including Bax, Bcl-2, GADD153, calregulin, and phosphorylated JNK, IRE1alpha, and eIF2alpha; all were ablated by the catalase transgene.	Paraquat	0-8	B cell leukemia/lymphoma 2	Mus musculus	142-147	
9124312-0	1997	Bcl-2 inhibits selective oxidation and externalization of phosphatidylserine during paraquat-induced apoptosis.	Paraquat	84-92	B cell leukemia/lymphoma 2	Mus musculus	0-5	
9681465-7	1998	Splenocytes from Bcl-2-deficient mice were also killed more effectively by dopamine as well as paraquat.	Paraquat	95-103	B cell leukemia/lymphoma 2	Mus musculus	17-22	
9124312-5	1997	Overexpression of Bcl-2 afforded significant protection against paraquat-induced apoptosis, early PS and PI oxidation, and PS externalization but not the ultimate formation of high-molecular-weight DNA fragments.	Paraquat	64-72	B cell leukemia/lymphoma 2	Mus musculus	18-23	
25111661-6	2014	PQ also induced more apoptosis in pneumocytes from MT(-/-) mice, and the expressions of apoptosis-related proteins Bax, Bcl-2, cleaved-caspase-3, and the ratio of Bax/Bcl-2 were all more significantly increased in PQ-treated MT(-/-) mice.	Paraquat	0-2	B cell leukemia/lymphoma 2	Mus musculus	120-125	
28115273-9	2017	Paraquat triggered O2- production, lipid peroxidation and apoptosis as evidenced by increased DHE staining, 4-HNE, caspase-3 activity, Bax and reduced Bcl-2 levels in association with unchanged ER stress.	Paraquat	0-8	B cell leukemia/lymphoma 2	Mus musculus	151-156	
28480221-6	2017	PQ exposure induced upregulation of the proapoptotic gene Bax and downregulation of the antiapoptotic gene Bcl-2, leading to marked cell apoptosis in the lung tissues.	Paraquat	0-2	B cell leukemia/lymphoma 2	Mus musculus	107-112	
31911963-10	2019	However, the co-administration of CCN and PQ significantly (p < 0.01, p < 0.01, p < 0.02) ameliorated the spermatogenesis ratio, upregulated the SOD level as well as bcl-2 expression, and reduced the MDA content and apoptosis vs the PQ-sole group.	Paraquat	42-44	B cell leukemia/lymphoma 2	Mus musculus	175-180	
30171970-8	2018	In addition, PQ reduced cellular beta-catenin, p-GSK-3beta, and cyclin-D1 and increased the radio of Bax/Bcl2.	Paraquat	13-15	B cell leukemia/lymphoma 2	Mus musculus	105-109	
29201275-11	2017	Furthermore, Res and/or MK reversed PQ-induced apoptosis assessed by differential expression of p53, Bax, and Bcl-2.	Paraquat	36-38	B cell leukemia/lymphoma 2	Mus musculus	110-115	
25111661-6	2014	PQ also induced more apoptosis in pneumocytes from MT(-/-) mice, and the expressions of apoptosis-related proteins Bax, Bcl-2, cleaved-caspase-3, and the ratio of Bax/Bcl-2 were all more significantly increased in PQ-treated MT(-/-) mice.	Paraquat	0-2	B cell leukemia/lymphoma 2	Mus musculus	167-172