PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20570573-9	2010	Upon further analysis, PA-infected patients who were treated with azithromycin had the highest arginase activity and the highest number of macrophages that were MR+TLR4-, and both of these markers were inversely related to the FEV(1).	Azithromycin	66-78	mannose receptor C-type 1	Homo sapiens	161-163	
30348950-9	2018	Azithromycin showed the same effect in imitated SLE macrophages, with distinct Akt phosphorylation at 30 min and 12 h. After inhibiting Akt phosphorylation by LY294002, the down-regulation of CD80, IL-1beta, IL-6, and TNF-alpha caused by azithromycin raised again, meanwhile, the up-regulation of CD206, Arg-1, Fizz-1, and IL-10 due to azithromycin was abolished.	Azithromycin	0-12	mannose receptor C-type 1	Homo sapiens	297-302	
18420960-0	2008	Azithromycin improves macrophage phagocytic function and expression of mannose receptor in chronic obstructive pulmonary disease.	Azithromycin	0-12	mannose receptor C-type 1	Homo sapiens	71-87	
18420960-4	2008	METHODS: In vitro effects of azithromycin on AM expression of MR were investigated.	Azithromycin	29-41	mannose receptor C-type 1	Homo sapiens	62-64	
18420960-10	2008	In patients with COPD, after azithromycin therapy, we observed significantly improved AM phagocytic ability (pre: 9.9%; post: 15.1%), reduced bronchial epithelial cell apoptosis (pre: 30.0%; post: 19.7%), and increased MR and reduced inflammatory markers in the peripheral blood.	Azithromycin	29-41	mannose receptor C-type 1	Homo sapiens	219-221	
18420960-11	2008	These findings implicate the MR in the defective phagocytic function of AMs in COPD and as a target for the azithromycin-mediated improvement in phagocytic ability.	Azithromycin	108-120	mannose receptor C-type 1	Homo sapiens	29-31