PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12381921-2	2002	Using whole kidney RNA from Aprt knockout mice, we previously showed that the renal deposition of DHA leads to changes in the expression of genes involved in tissue injury.	2,8-dihydroxyadenine	98-101	adenine phosphoribosyl transferase	Mus musculus	28-32	
11532086-20	2001	CONCLUSIONS: Both male and female Aprt knockout mice accumulate DHA.	2,8-dihydroxyadenine	64-67	adenine phosphoribosyl transferase	Mus musculus	34-38	
9689017-3	1998	In the absence of APRT, 2,8-dihydroxyadenine (DHA) is produced from adenine by xanthine dehydrogenase (XDH) and can precipitate in the renal interstitium, resulting in kidney disease.	2,8-dihydroxyadenine	24-44	adenine phosphoribosyl transferase	Mus musculus	18-22	
8864750-9	1996	This has proved an effective therapy for APRT null mice, preventing accumulation of 2,8-dihydroxyadenine and much of the resultant renal obstruction, allowing us to establish a breeding line.	2,8-dihydroxyadenine	84-104	adenine phosphoribosyl transferase	Mus musculus	41-45	
8643571-3	1996	Mice homozygous for a null Aprt allele excrete adenine and DHA crystals in the urine.	2,8-dihydroxyadenine	59-62	adenine phosphoribosyl transferase	Mus musculus	27-31