PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27222100-0	2016	Increased expression and activity of p75NTR are crucial events in azacitidine-induced cell death in prostate cancer.	Azacitidine	66-77	nerve growth factor receptor	Homo sapiens	37-43	
27222100-3	2016	We aimed to verify the role of p75NTR in the azacitidine-mediated antitumor effects on 22Rv1 and PC3 androgen-independent prostate cancer cells.	Azacitidine	45-56	nerve growth factor receptor	Homo sapiens	31-37	
27222100-4	2016	In the present study, we reported that the antiproliferative and pro-apoptotic effects of 5-azacytidine (azacitidine) were more marked in the presence of physiological concentrations of NGF and were reduced when a blocking p75NTR antibody or the selective p75NTR inhibitor, Ro 08-2750, were used.	Azacitidine	90-103	nerve growth factor receptor	Homo sapiens	223-229	
27222100-4	2016	In the present study, we reported that the antiproliferative and pro-apoptotic effects of 5-azacytidine (azacitidine) were more marked in the presence of physiological concentrations of NGF and were reduced when a blocking p75NTR antibody or the selective p75NTR inhibitor, Ro 08-2750, were used.	Azacitidine	90-103	nerve growth factor receptor	Homo sapiens	256-262	
27222100-4	2016	In the present study, we reported that the antiproliferative and pro-apoptotic effects of 5-azacytidine (azacitidine) were more marked in the presence of physiological concentrations of NGF and were reduced when a blocking p75NTR antibody or the selective p75NTR inhibitor, Ro 08-2750, were used.	Azacitidine	105-116	nerve growth factor receptor	Homo sapiens	223-229	
27222100-4	2016	In the present study, we reported that the antiproliferative and pro-apoptotic effects of 5-azacytidine (azacitidine) were more marked in the presence of physiological concentrations of NGF and were reduced when a blocking p75NTR antibody or the selective p75NTR inhibitor, Ro 08-2750, were used.	Azacitidine	105-116	nerve growth factor receptor	Homo sapiens	256-262	
27222100-5	2016	Azacitidine increased the expression of p75NTR without interfering with the expression of the low affinity NGF receptor TrkA and induced caspase 9-dependent caspase 3 activity.	Azacitidine	0-11	nerve growth factor receptor	Homo sapiens	40-46	
21475909-0	2009	Estrogen in combination with 5-azacitidine up-regulates p75NTR expression and induces apoptosis in 22Rv1 prostate cancer cells.	Azacitidine	29-42	nerve growth factor receptor	Homo sapiens	56-62	
21475909-5	2009	These results provide evidence that the up-regulation of ESR2 and p75NTR by estrogen plus 5-AzaC may be a potential therapeutic strategy for the treatment of androgen-refractory prostate cancer.	Azacitidine	90-96	nerve growth factor receptor	Homo sapiens	66-72