PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27404495-9	2016	However, in other cell lines without GSTM1, 5-aza-dC increased GSTM5 expression and decreased its CpG DNA methylation ratio from 84.6% to 61.5% in 5637, and from 97.4% to 75% in J82 cells.	Azacitidine	44-49	glutathione S-transferase mu 5	Homo sapiens	63-68	
33802702-1	2021	Our previous study demonstrated that the glutathione S-transferase Mu 5 (GSTM5) gene is highly CpG-methylated in bladder cancer cells and that demethylation by 5-aza-dC activates GSTM5 gene expression.	Azacitidine	160-165	glutathione S-transferase mu 5	Homo sapiens	41-71	
33802702-1	2021	Our previous study demonstrated that the glutathione S-transferase Mu 5 (GSTM5) gene is highly CpG-methylated in bladder cancer cells and that demethylation by 5-aza-dC activates GSTM5 gene expression.	Azacitidine	160-165	glutathione S-transferase mu 5	Homo sapiens	73-78	
33802702-1	2021	Our previous study demonstrated that the glutathione S-transferase Mu 5 (GSTM5) gene is highly CpG-methylated in bladder cancer cells and that demethylation by 5-aza-dC activates GSTM5 gene expression.	Azacitidine	160-165	glutathione S-transferase mu 5	Homo sapiens	179-184