PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32503264-13	2020	Accordingly, the expression levels of Rasal1 and Pten, which are antifibrotic genes, were increased by treatment after the 5-aza-dC and combined treatments.	Azacitidine	123-128	phosphatase and tensin homolog	Rattus norvegicus	49-53	
24680972-12	2014	PTEN mRNA expression was up regulated and PTEN methylation level was reduced by the epigenetic inhibitors, 5-aza and TSA.	Azacitidine	107-112	phosphatase and tensin homolog	Rattus norvegicus	0-4	
31085236-11	2019	Furthermore, the demethylation of PTEN, which was induced by 5-Aza-dC, inhibited the proliferation, migration and promoted apoptosis in PASMCs.	Azacitidine	61-66	phosphatase and tensin homolog	Rattus norvegicus	34-38	
31085236-12	2019	In vivo studies further demonstrated that the expression of PTEN, mean pulmonary artery pressure and right ventricular hypertrophy index in HPH rats was attenuated by 5-Aza-dC.	Azacitidine	167-172	phosphatase and tensin homolog	Rattus norvegicus	60-64	
31085236-13	2019	5-Aza-dC also suppressed the expression of DNMTs and PTEN methylation in the lungs of HPH rats.	Azacitidine	0-5	phosphatase and tensin homolog	Rattus norvegicus	53-57	
24680972-12	2014	PTEN mRNA expression was up regulated and PTEN methylation level was reduced by the epigenetic inhibitors, 5-aza and TSA.	Azacitidine	107-112	phosphatase and tensin homolog	Rattus norvegicus	42-46