PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15620579-8	2005	After 5-Aza treatment, these cells expressed TbetaR-II at both mRNA and protein levels, which corresponded to the restoration of sensitivity to TGF-beta by an increase in P-Smad2.	Azacitidine	6-11	transforming growth factor, beta 1	Mus musculus	144-152	
22457795-8	2012	At the 48(th) week after Cd exposure, livers from Cd-treated mice displayed the increased caspase-8 CGI methylation and decreased caspase-8 protein expression, along with significant increases in cell proliferation and overexpression of TGF-beta1 and cytokeratin 8/18 (the latter is a new marker of mouse liver preneoplastic lesions), all which were prevented by 5-aza treatment.	Azacitidine	363-368	transforming growth factor, beta 1	Mus musculus	237-246	
35523401-12	2022	Moreover, treatment of HK-2 cells with 5-aza-dc reversed the activation of the TGF-beta1-induced PI3K/AKT signalling pathway, which inhibited renal fibrosis.	Azacitidine	39-44	transforming growth factor, beta 1	Mus musculus	79-88	
26731474-6	2016	Mechanistically, the profibrotic factor TGF-beta1 induced hypermethylation and repression of erythropoietin in pericytes; these effects were prevented by 5-azacytidine treatment.	Azacitidine	154-167	transforming growth factor, beta 1	Mus musculus	40-49