PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15371442-7	2004	The loss of Cx43 protein was counteracted by ammonium chloride, indicating that acidification of internalized Cx43 gap junctions is a prerequisite for its degradation.	Ammonium Chloride	45-62	gap junction protein, alpha 1	Rattus norvegicus	12-16	
16633735-4	2006	Such abnormalities in the expression of Cx43 were alleviated by treatment with either lysosomal (NH(4)Cl, Leupeptin) or proteasomal inhibitor (ALLN).	Ammonium Chloride	97-104	gap junction protein, alpha 1	Rattus norvegicus	40-44	
15371442-7	2004	The loss of Cx43 protein was counteracted by ammonium chloride, indicating that acidification of internalized Cx43 gap junctions is a prerequisite for its degradation.	Ammonium Chloride	45-62	gap junction protein, alpha 1	Rattus norvegicus	110-114	
9747439-5	1998	RESULTS: Immunoblots of whole cell lysates demonstrated increased levels of connexin43 in cultures treated with lysosomal inhibitors (chloroquine, leupeptin, E-64, or ammonium chloride) or proteasomal inhibitors (lactacystin or ALLN).	Ammonium Chloride	167-184	gap junction protein, alpha 1	Rattus norvegicus	76-86	
23403203-5	2013	Inhibition of lysosomes or proteasomes by co-treatment with antofine and their respective specific inhibitors, NH4Cl or MG132, partially inhibited the antofine-induced decrease in Cx43 protein levels, but did not inhibit the antofine-induced inhibition of GJIC.	Ammonium Chloride	111-116	gap junction protein, alpha 1	Rattus norvegicus	180-184	
33765917-12	2021	The Cx43 expression was slightly increased in M30 co-cultures after 6 h incubation with 5 mM NH4Cl.	Ammonium Chloride	93-98	gap junction protein, alpha 1	Rattus norvegicus	4-8	
33765917-13	2021	Also, the AQP4 expression was slightly increased only in M5 co-cultures treated with 10 mM NH4Cl for 6 h. Under the other conditions, Cx43 and AQP4 expression was not affected by NH4Cl.	Ammonium Chloride	91-96	gap junction protein, alpha 1	Rattus norvegicus	134-138