PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12623784-1	2003	Carbon monoxide (CO) is produced from heme by heme oxygenase-2 (HO-2) in cerebral blood vessels.	Carbon Monoxide	0-15	heme oxygenase 2	Sus scrofa	46-62	
12623784-1	2003	Carbon monoxide (CO) is produced from heme by heme oxygenase-2 (HO-2) in cerebral blood vessels.	Carbon Monoxide	0-15	heme oxygenase 2	Sus scrofa	64-68	
12623784-1	2003	Carbon monoxide (CO) is produced from heme by heme oxygenase-2 (HO-2) in cerebral blood vessels.	Carbon Monoxide	17-19	heme oxygenase 2	Sus scrofa	46-62	
12623784-1	2003	Carbon monoxide (CO) is produced from heme by heme oxygenase-2 (HO-2) in cerebral blood vessels.	Carbon Monoxide	17-19	heme oxygenase 2	Sus scrofa	64-68	
35203567-1	2022	Carbon monoxide (CO) has been proposed as a chemical light signal and neural system modulator via heme oxygenases -1 and -2 (HO-1 and HO-2).	Carbon Monoxide	0-15	heme oxygenase 2	Sus scrofa	134-138	
35203567-1	2022	Carbon monoxide (CO) has been proposed as a chemical light signal and neural system modulator via heme oxygenases -1 and -2 (HO-1 and HO-2).	Carbon Monoxide	17-19	heme oxygenase 2	Sus scrofa	134-138	
19118162-12	2009	The ability of CO and bilirubin to combat TNF-alpha-induced oxidative stress by inhibiting Nox4 activity and/or by O(2)(*-) scavenging, taken together with close intracellular compartmentalization of HO-2 and Nox4 in cerebral vascular endothelium, may contribute to HO-2 cytoprotection against inflammatory cerebrovascular disease.	Carbon Monoxide	15-17	heme oxygenase 2	Sus scrofa	200-204	
19118162-12	2009	The ability of CO and bilirubin to combat TNF-alpha-induced oxidative stress by inhibiting Nox4 activity and/or by O(2)(*-) scavenging, taken together with close intracellular compartmentalization of HO-2 and Nox4 in cerebral vascular endothelium, may contribute to HO-2 cytoprotection against inflammatory cerebrovascular disease.	Carbon Monoxide	15-17	heme oxygenase 2	Sus scrofa	266-270