PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3118370-5	1987	A large part, about one-third, of the steady-state oxygen uptake is abolished by carbon monoxide blockade of myoglobin oxygenation.	Carbon Monoxide	81-96	myoglobin	Rattus norvegicus	109-118	
551635-0	1979	Effect of repeated carbon monoxide intoxications on the myoglobin concentration in heart and skeletal muscle of rats.	Carbon Monoxide	19-34	myoglobin	Rattus norvegicus	56-65	
8155298-1	1993	By sequestering intracellular myoglobin of cardiac muscle cells in the nonfunctioning carboxymyoglobin form, carbon monoxide blocks myoglobin-facilitated diffusion of oxygen, as well as myoglobin-mediated oxidative phosphorylation.	Carbon Monoxide	109-124	myoglobin	Rattus norvegicus	30-39	
8155298-1	1993	By sequestering intracellular myoglobin of cardiac muscle cells in the nonfunctioning carboxymyoglobin form, carbon monoxide blocks myoglobin-facilitated diffusion of oxygen, as well as myoglobin-mediated oxidative phosphorylation.	Carbon Monoxide	109-124	myoglobin	Rattus norvegicus	93-102	
8155298-1	1993	By sequestering intracellular myoglobin of cardiac muscle cells in the nonfunctioning carboxymyoglobin form, carbon monoxide blocks myoglobin-facilitated diffusion of oxygen, as well as myoglobin-mediated oxidative phosphorylation.	Carbon Monoxide	109-124	myoglobin	Rattus norvegicus	93-102	
8155298-2	1993	Here, we explore the hypothesis that the carbon monoxide blockade of myoglobin function may be responsible at the cellular level for a component of the cardiotoxicity of carbon monoxide observed during exercise.	Carbon Monoxide	41-56	myoglobin	Rattus norvegicus	69-78	
8155298-2	1993	Here, we explore the hypothesis that the carbon monoxide blockade of myoglobin function may be responsible at the cellular level for a component of the cardiotoxicity of carbon monoxide observed during exercise.	Carbon Monoxide	170-185	myoglobin	Rattus norvegicus	69-78	
8155298-10	1993	When carboxymyoglobin was at least 40% of the total intracellular myoglobin, we found that the ratio of phosphocreatine to adenosine triphosphate in carbon monoxide-treated heart cells was significantly lower than that in control cells from the same preparation.	Carbon Monoxide	149-164	myoglobin	Rattus norvegicus	12-21