PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1234703-0	1975	Extraction of biologically active erythropoietin from kidneys of carbon monoxide intoxicated mice.	Carbon Monoxide	65-80	erythropoietin	Mus musculus	34-48	
854648-0	1977	Influence of ambient temperature on erythropoietin production in carbon monoxide-intoxicated mice.	Carbon Monoxide	65-80	erythropoietin	Mus musculus	36-50	
935624-1	1976	The effects of the beta-adrenergic blocking agent (propranolol) on erythropoietin production in carbon monoxide intoxicated mice were studied.	Carbon Monoxide	96-111	erythropoietin	Mus musculus	67-81	
935624-2	1976	The raise in plasma erythropoietin after a 4 hour intoxication with carbon monoxide was 47% less in the propranolol treated group.	Carbon Monoxide	68-83	erythropoietin	Mus musculus	20-34	
1234703-2	1975	From 2 to 4 hr after the start of carbon monoxide (CO)-hypoxemia large amounts of erythropoietin (ESF) are released in the plasma.	Carbon Monoxide	34-49	erythropoietin	Mus musculus	82-96	
1234703-2	1975	From 2 to 4 hr after the start of carbon monoxide (CO)-hypoxemia large amounts of erythropoietin (ESF) are released in the plasma.	Carbon Monoxide	51-53	erythropoietin	Mus musculus	82-96	
10678740-3	2000	EPO gene expression was stimulated by normobaric hypoxia (8% O2) or by 0.1% carbon monoxide (CO) inhalation for 4 h each, or by intraperitoneal injection of 60 mg/kg cobaltous chloride (CoCl2) for 6 h. Renal EPO mRNA in wt increased 12-, 40-, and 13-fold over normoxic levels in response to hypoxia, CO and CoCl2 respectively.	Carbon Monoxide	76-91	erythropoietin	Mus musculus	0-3	
10678740-3	2000	EPO gene expression was stimulated by normobaric hypoxia (8% O2) or by 0.1% carbon monoxide (CO) inhalation for 4 h each, or by intraperitoneal injection of 60 mg/kg cobaltous chloride (CoCl2) for 6 h. Renal EPO mRNA in wt increased 12-, 40-, and 13-fold over normoxic levels in response to hypoxia, CO and CoCl2 respectively.	Carbon Monoxide	93-95	erythropoietin	Mus musculus	0-3	
10678740-3	2000	EPO gene expression was stimulated by normobaric hypoxia (8% O2) or by 0.1% carbon monoxide (CO) inhalation for 4 h each, or by intraperitoneal injection of 60 mg/kg cobaltous chloride (CoCl2) for 6 h. Renal EPO mRNA in wt increased 12-, 40-, and 13-fold over normoxic levels in response to hypoxia, CO and CoCl2 respectively.	Carbon Monoxide	300-302	erythropoietin	Mus musculus	0-3	
2316714-3	1990	We found that EPO formation in mice exposed to normobaric hypoxia (8% O2) or to carbon monoxide (0.1%) was reduced by 30 and 42% when animals were simultaneously exposed to hypercapnia (7% CO2), by 35 and 38% when subjected to metabolic acidosis (NH4Cl), and unchanged when subjected to metabolic alkalosis (NaHCO3).	Carbon Monoxide	80-95	erythropoietin	Mus musculus	14-17	
7731971-5	1995	Exposure to 0.1% carbon monoxide, a procedure that causes functional anemia, resulted in a 20-fold increase of EPO mRNA in mouse brain as quantified by competitive reverse transcription-PCR, whereas the EPO-R mRNA level was not influenced by hypoxia.	Carbon Monoxide	17-32	erythropoietin	Mus musculus	111-114