PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24505052-1	2014	The main purpose of this study was to assess the role of S100B protein, neuron-specific enolase (NSE), and glial fibrillary acidic protein (GFAP) in the evaluation of hypoxic brain injury in acute carbon monoxide (CO)-poisoned patients.	Carbon Monoxide	197-212	enolase 2	Homo sapiens	72-95	
26040109-0	2015	Serum neuron-specific enolase and S-100beta levels as prognostic follow-up markers for oxygen administered carbon monoxide intoxication cases.	Carbon Monoxide	107-122	enolase 2	Homo sapiens	6-29	
15025610-3	2004	We hypothesized that increased serum concentrations of Neuron-specific enolase (NSE) and S-100beta protein could be detected after carbon monoxide poisoning and that the concentration would correlate with the severity of intoxication.	Carbon Monoxide	131-146	enolase 2	Homo sapiens	55-78	
23380091-0	2013	Neuron-specific enolase and S100B protein in children with carbon monoxide poisoning: children are not just small adults.	Carbon Monoxide	59-74	enolase 2	Homo sapiens	0-23	
23380091-1	2013	INTRODUCTION: The aim of this study was to evaluate the role of S100B protein and neuron-specific enolase (NSE) in children with carbon monoxide (CO) poisoning.	Carbon Monoxide	129-144	enolase 2	Homo sapiens	82-105	
23380091-1	2013	INTRODUCTION: The aim of this study was to evaluate the role of S100B protein and neuron-specific enolase (NSE) in children with carbon monoxide (CO) poisoning.	Carbon Monoxide	129-144	enolase 2	Homo sapiens	107-110	
23380091-1	2013	INTRODUCTION: The aim of this study was to evaluate the role of S100B protein and neuron-specific enolase (NSE) in children with carbon monoxide (CO) poisoning.	Carbon Monoxide	146-148	enolase 2	Homo sapiens	82-105	
23380091-1	2013	INTRODUCTION: The aim of this study was to evaluate the role of S100B protein and neuron-specific enolase (NSE) in children with carbon monoxide (CO) poisoning.	Carbon Monoxide	146-148	enolase 2	Homo sapiens	107-110	
20006203-0	2010	S-100beta and neuron-specific enolase levels in carbon monoxide-related brain injury.	Carbon Monoxide	48-63	enolase 2	Homo sapiens	14-37	
19683113-0	2009	Elevated serum S100B protein and neuron-specific enolase levels in carbon monoxide poisoning.	Carbon Monoxide	67-82	enolase 2	Homo sapiens	33-56	
15025610-3	2004	We hypothesized that increased serum concentrations of Neuron-specific enolase (NSE) and S-100beta protein could be detected after carbon monoxide poisoning and that the concentration would correlate with the severity of intoxication.	Carbon Monoxide	131-146	enolase 2	Homo sapiens	80-83	
34533065-0	2021	Prediction of delayed neuropsychiatric sequelae after carbon monoxide poisoning via serial determination of serum neuron-specific enolase levels.	Carbon Monoxide	54-69	enolase 2	Homo sapiens	114-137	
9842417-5	1998	Cases of carbon monoxide intoxication by burning showed a higher incidence of NSE immunoreactivity in the cell nucleus of the XII than other cases, while there was no statistically significant correlation between NSE immunoreactivity in the cell nucleus and the Nissl amount.	Carbon Monoxide	9-24	enolase 2	Homo sapiens	78-81	
33984366-0	2021	Neuropsychiatric sequelae of acute carbon monoxide poisoning: The predictive role of neuron specific enolase and glial fibrillary acidic protein.	Carbon Monoxide	35-50	enolase 2	Homo sapiens	85-108	
33123300-0	2020	Association between Neuron-Specific Enolase Gene Polymorphism and Delayed Encephalopathy after Acute Carbon Monoxide Poisoning.	Carbon Monoxide	101-116	enolase 2	Homo sapiens	20-43	
33123300-1	2020	Objective: The aim of this study is to explore the relationship between neuron-specific enolase (NSE) gene polymorphism and delayed encephalopathy after acute carbon monoxide poisoning (DEACMP) and provide a theoretical basis for DEACMP pathogenesis, diagnosis, and prognosis.	Carbon Monoxide	159-174	enolase 2	Homo sapiens	72-95	
33123300-1	2020	Objective: The aim of this study is to explore the relationship between neuron-specific enolase (NSE) gene polymorphism and delayed encephalopathy after acute carbon monoxide poisoning (DEACMP) and provide a theoretical basis for DEACMP pathogenesis, diagnosis, and prognosis.	Carbon Monoxide	159-174	enolase 2	Homo sapiens	97-100	
28349731-0	2018	Serum neuron-specific enolase as an early predictor of delayed neuropsychiatric sequelae in patients with acute carbon monoxide poisoning.	Carbon Monoxide	112-127	enolase 2	Homo sapiens	6-29	
29239210-0	2018	Serum neuron-specific enolase levels at presentation and long-term neurological sequelae after acute charcoal burning-induced carbon monoxide poisoning.	Carbon Monoxide	126-141	enolase 2	Homo sapiens	6-29	
29239210-1	2018	OBJECTIVE: This study aimed to investigate whether clinical parameters and serum neuron-specific enolase (NSE) levels measured at emergency department (ED) presentation help stratify the risk of acute or delayed persistent severe neurological sequelae after acute carbon monoxide (CO) poisoning induced by charcoal burning.	Carbon Monoxide	264-279	enolase 2	Homo sapiens	106-109	
29239210-1	2018	OBJECTIVE: This study aimed to investigate whether clinical parameters and serum neuron-specific enolase (NSE) levels measured at emergency department (ED) presentation help stratify the risk of acute or delayed persistent severe neurological sequelae after acute carbon monoxide (CO) poisoning induced by charcoal burning.	Carbon Monoxide	281-283	enolase 2	Homo sapiens	106-109