PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 20674229-1 2011 It is proposed that the significant elevation of interleukin-6 (>400 pg/mL) in cerebrospinal fluid during the early phase of carbon monoxide poisoning may be a predictive biomarker for the development of delayed encephalopathy. Carbon Monoxide 128-143 interleukin 6 Homo sapiens 49-62 20674229-6 2011 Interleukin-6 in cerebrospinal fluid at 5.5 hours after his last exposure to carbon monoxide was significantly elevated (752 pg/mL). Carbon Monoxide 77-92 interleukin 6 Homo sapiens 0-13 19857421-1 2009 OBJECTIVE: This study was designed to investigate whether interleukin 6 (IL-6) in cerebrospinal fluid (CSF) in the early phase of carbon monoxide (CO) poisoning can be a predictive marker of delayed encephalopathy (DE). Carbon Monoxide 130-145 interleukin 6 Homo sapiens 58-71 19857421-1 2009 OBJECTIVE: This study was designed to investigate whether interleukin 6 (IL-6) in cerebrospinal fluid (CSF) in the early phase of carbon monoxide (CO) poisoning can be a predictive marker of delayed encephalopathy (DE). Carbon Monoxide 130-145 interleukin 6 Homo sapiens 73-77 19857421-1 2009 OBJECTIVE: This study was designed to investigate whether interleukin 6 (IL-6) in cerebrospinal fluid (CSF) in the early phase of carbon monoxide (CO) poisoning can be a predictive marker of delayed encephalopathy (DE). Carbon Monoxide 147-149 interleukin 6 Homo sapiens 58-71 19857421-1 2009 OBJECTIVE: This study was designed to investigate whether interleukin 6 (IL-6) in cerebrospinal fluid (CSF) in the early phase of carbon monoxide (CO) poisoning can be a predictive marker of delayed encephalopathy (DE). Carbon Monoxide 147-149 interleukin 6 Homo sapiens 73-77 12762340-4 2003 The possibility that exhaled IL-6 levels are related to exhaled carbon monoxide (CO) and lung function has also been explored. Carbon Monoxide 64-79 interleukin 6 Homo sapiens 29-33 17339835-1 2007 In this issue of British Journal of Pharmacology, Megias and colleagues demonstrate how preincubation of human colonic Caco-2 cells with CORM-2, a carbon monoxide releasing molecule (CO-RM), reduces the expression of inducible nitric oxide synthase, interleukin (IL)-6 and IL-8 caused by proinflammatory cytokines. Carbon Monoxide 147-162 interleukin 6 Homo sapiens 250-268 16405788-0 2005 [Change of the serum interleukin 6 in patients with delayed encephalopathy after acute carbon monoxide poisoning]. Carbon Monoxide 87-102 interleukin 6 Homo sapiens 21-34 16003000-0 2005 Carbon monoxide inhibits IL-17-induced IL-6 production through the MAPK pathway in human pulmonary epithelial cells. Carbon Monoxide 0-15 interleukin 6 Homo sapiens 39-43 16126982-9 2005 IL-6 levels correlated with the leukocyte count, sIL-2r with the FQ, and ET-1 with the diffuse lung capacity for carbon monoxide. Carbon Monoxide 113-128 interleukin 6 Homo sapiens 0-4 19750100-7 2009 RESULTS: We found the most consistent modifications for variants in IL6 rs2069832 and FBG rs1800790 after exposure to carbon monoxide (CO; 24-hr average; p-values for interaction, 0.034 and 0.019, respectively). Carbon Monoxide 118-133 interleukin 6 Homo sapiens 68-71 19750100-7 2009 RESULTS: We found the most consistent modifications for variants in IL6 rs2069832 and FBG rs1800790 after exposure to carbon monoxide (CO; 24-hr average; p-values for interaction, 0.034 and 0.019, respectively). Carbon Monoxide 135-137 interleukin 6 Homo sapiens 68-71 18332643-3 2008 OBJECTIVES: We hypothesize that end-tidal carbon monoxide corrected for inhaled CO (ETCOc), malondialdehyde (MDA) (markers of oxidative stress) and proinflammatory cytokine (IL-6, IL-8) production are higher in infants of preeclamptic mothers with HELLP syndrome than in those of preeclamptic mothers without HELLP syndrome. Carbon Monoxide 42-57 interleukin 6 Homo sapiens 174-178 16572448-8 2006 When hemoglobin, as a scavenger of carbon monoxide, was added to auranofin-treated synovial cell lines, LPS-dependent production of IL-6 and IL-8 was increased. Carbon Monoxide 35-50 interleukin 6 Homo sapiens 132-136 12762340-4 2003 The possibility that exhaled IL-6 levels are related to exhaled carbon monoxide (CO) and lung function has also been explored. Carbon Monoxide 81-83 interleukin 6 Homo sapiens 29-33 35218740-0 2022 Carbon monoxide releasing molecule-2 attenuates angiotensin II-induced IL-6/Jak2/Stat3-associated inflammation by inhibiting NADPH oxidase- and mitochondria-derived ROS in human aortic smooth muscle cells. Carbon Monoxide 0-15 interleukin 6 Homo sapiens 71-75 34336088-0 2021 Carbon Monoxide-Releasing Molecule-2 Ameliorates Particulate Matter-Induced Aorta Inflammation via Toll-Like Receptor/NADPH Oxidase/ROS/NF-kappaB/IL-6 Inhibition. Carbon Monoxide 0-15 interleukin 6 Homo sapiens 146-150 11371414-9 2001 Moreover, using impairment of carbon monoxide transfer (DL(CO)) adjusted for duration of dyspnea as a marker of rapidity of disease progression, we found that the IL-6 intron 4GG genotype was the only genotype independently associated with lower DL(CO) levels. Carbon Monoxide 30-45 interleukin 6 Homo sapiens 163-167