PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15181156-4	2004	Five genes were found that are known to be involved in pathways of detoxification of peroxide (catalase, glutathione peroxidase-1, peroxiredoxin-1, peroxiredoxin-5, and nuclear factor erythroid-derived 2-like 2).	Peroxides	85-93	catalase	Mus musculus	95-103	
4019779-10	1985	The observed suppression of two different types of inflammation (an immune and a nonimmune arthritis) by catalase and peroxidase suggests that elimination of peroxides contributes to the suppression of an inflammatory response.	Peroxides	158-167	catalase	Mus musculus	105-113	
19969073-5	2010	Additional experiments with mice with disrupted catalase or glutathione peroxidase (Gpx1) genes showed that Gpx1 is the only erythrocyte enzyme that reduces organic peroxides.	Peroxides	165-174	catalase	Mus musculus	48-56	
16413415-10	2006	The overall results suggest that enriching the cellular CAT gene level with an appropriate recombinant viral vector may be sufficient to protect in vivo systems from peroxide stress.	Peroxides	166-174	catalase	Mus musculus	56-59	
15050730-5	2004	The EPE-induced translocation of AIF was suppressed with the addition of catalase, suggesting that the rapid intracellular peroxide levels after addition of EPE triggers off induction of apoptosis, which is AIF-mediated and caspase-independent.	Peroxides	123-131	catalase	Mus musculus	73-81	
14609559-1	2003	Immortal murine lens epithelial cells which were conditioned to survive peroxide stress were found to have a remarkable increase in catalase activity as well as lesser changes in a number of other antioxidative defense systems [Invest.	Peroxides	72-80	catalase	Mus musculus	132-140	
14609559-8	2003	It was found that inhibition of catalase caused peroxide resistant cells to die within 48-72 hr when exposed to normally tolerated concentrations of peroxide.	Peroxides	48-56	catalase	Mus musculus	32-40	
14609559-8	2003	It was found that inhibition of catalase caused peroxide resistant cells to die within 48-72 hr when exposed to normally tolerated concentrations of peroxide.	Peroxides	149-157	catalase	Mus musculus	32-40	
14580681-8	2003	The EPE-induced translocation of AIF was suppressed with the addition of catalase, suggesting that the rapid intracellular peroxide levels after addition of EPE triggers off induction of apoptosis, which is AIF-mediated and caspase-independent.	Peroxides	123-131	catalase	Mus musculus	73-81	
8913362-5	1996	Apoptosis seems to be produced by dopamine autoxidation, because intracellular peroxides increase after dopamine treatment and cell death can be inhibited by catalase and N-acetylcysteine.	Peroxides	79-88	catalase	Mus musculus	158-166	
34598016-4	2021	Fortunately, several mechanisms exist to buffer bursts of intracellular ROS and peroxide production, including the enzymes Catalase, Glutathione Peroxidase and Superoxide Dismutase (SOD).	Peroxides	80-88	catalase	Mus musculus	123-131