PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 16413415-10 2006 The overall results suggest that enriching the cellular CAT gene level with an appropriate recombinant viral vector may be sufficient to protect in vivo systems from peroxide stress. Peroxides 166-174 catalase Mus musculus 56-59 19969073-5 2010 Additional experiments with mice with disrupted catalase or glutathione peroxidase (Gpx1) genes showed that Gpx1 is the only erythrocyte enzyme that reduces organic peroxides. Peroxides 165-174 catalase Mus musculus 48-56 8913362-5 1996 Apoptosis seems to be produced by dopamine autoxidation, because intracellular peroxides increase after dopamine treatment and cell death can be inhibited by catalase and N-acetylcysteine. Peroxides 79-88 catalase Mus musculus 158-166 15181156-4 2004 Five genes were found that are known to be involved in pathways of detoxification of peroxide (catalase, glutathione peroxidase-1, peroxiredoxin-1, peroxiredoxin-5, and nuclear factor erythroid-derived 2-like 2). Peroxides 85-93 catalase Mus musculus 95-103 14580681-8 2003 The EPE-induced translocation of AIF was suppressed with the addition of catalase, suggesting that the rapid intracellular peroxide levels after addition of EPE triggers off induction of apoptosis, which is AIF-mediated and caspase-independent. Peroxides 123-131 catalase Mus musculus 73-81 15050730-5 2004 The EPE-induced translocation of AIF was suppressed with the addition of catalase, suggesting that the rapid intracellular peroxide levels after addition of EPE triggers off induction of apoptosis, which is AIF-mediated and caspase-independent. Peroxides 123-131 catalase Mus musculus 73-81 14609559-1 2003 Immortal murine lens epithelial cells which were conditioned to survive peroxide stress were found to have a remarkable increase in catalase activity as well as lesser changes in a number of other antioxidative defense systems [Invest. Peroxides 72-80 catalase Mus musculus 132-140 14609559-8 2003 It was found that inhibition of catalase caused peroxide resistant cells to die within 48-72 hr when exposed to normally tolerated concentrations of peroxide. Peroxides 48-56 catalase Mus musculus 32-40 14609559-8 2003 It was found that inhibition of catalase caused peroxide resistant cells to die within 48-72 hr when exposed to normally tolerated concentrations of peroxide. Peroxides 149-157 catalase Mus musculus 32-40 4019779-10 1985 The observed suppression of two different types of inflammation (an immune and a nonimmune arthritis) by catalase and peroxidase suggests that elimination of peroxides contributes to the suppression of an inflammatory response. Peroxides 158-167 catalase Mus musculus 105-113 34598016-4 2021 Fortunately, several mechanisms exist to buffer bursts of intracellular ROS and peroxide production, including the enzymes Catalase, Glutathione Peroxidase and Superoxide Dismutase (SOD). Peroxides 80-88 catalase Mus musculus 123-131