PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3263331-3	1988	Lithium also caused an increase in interleukin 1 production from peripheral blood monocytes.	Lithium	0-7	interleukin 1 alpha	Homo sapiens	35-48	
9342951-7	1997	The products of Smith degradation (T2a-S) and lithium degradation (T2a-L) of T2a and the product of deacetylation (T2b-D) of T2b also induced monocytes to secret IL-1 as efficiently as the original polysaccharides, indicating that xylosyl and glucuronic acid residues as well as acetyl groups were not important to promote the cytokine-stimulating activity.	Lithium	46-53	interleukin 1 alpha	Homo sapiens	162-166	
2553837-3	1989	Since both tumor necrosis factor alpha (TNF alpha) and interleukin 1 (IL-1), which are secreted by monocytes, can stimulate endothelial cells to produce granulocyte-macrophage colony-stimulating factor (GM-CSF), we determined whether lithium-stimulated monocytes produced TNF alpha and/or IL-1.	Lithium	234-241	interleukin 1 alpha	Homo sapiens	55-74	
2553837-3	1989	Since both tumor necrosis factor alpha (TNF alpha) and interleukin 1 (IL-1), which are secreted by monocytes, can stimulate endothelial cells to produce granulocyte-macrophage colony-stimulating factor (GM-CSF), we determined whether lithium-stimulated monocytes produced TNF alpha and/or IL-1.	Lithium	234-241	interleukin 1 alpha	Homo sapiens	70-74