PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23587524-4	2013	ATRA induced upregulation of Pyk2 mRNA, protein and phosphorylation levels and enhanced Pyk2 interaction with paxillin and vinculin.	Tretinoin	0-4	vinculin	Homo sapiens	123-131	
6188622-0	1983	Formation of vinculin plaques precedes other cytoskeletal changes during retinoic acid-induced teratocarcinoma cell differentiation.	Tretinoin	73-86	vinculin	Homo sapiens	13-21	
6188622-8	1983	This sequence of changes suggests that the vinculin-containing adhesion plaques may be important in the mechanism of RA-induced differentiation of EC cells.	Tretinoin	117-119	vinculin	Homo sapiens	43-51	
3099098-7	1986	The changes in PK-C activity in TPA + RA-treated cells were accompanied by Ca2+/phospholipid(PL)-dependent phosphorylation in vitro of pp38 which is characteristic of treatment with RA alone, as well as the Ca2+/PL-independent phosphorylation in vitro of pp82 and pp130 (vinculin) which is prevalent in cells treated continuously with TPA alone and is absent in RA-treated cells.	Tretinoin	38-40	vinculin	Homo sapiens	271-279	
32254654-11	2018	Both cell types exhibited more vinculin expression when seeded to RA-loaded PCL scaffolds.	Tretinoin	66-68	vinculin	Homo sapiens	31-39	
1534272-6	1992	In contrast, cells exposed to RA contained larger amounts of alpha-actinin, vinculin, talin, lipocortin I, and lipocortin II, as determined with their respective antibodies followed by flow cytometric analysis as described above.	Tretinoin	30-32	vinculin	Homo sapiens	76-84	
1534272-8	1992	Treatment of HL-60/S4 with RA led to increases in vinculin, talin, lipocortin I, and lipocortin II.	Tretinoin	27-29	vinculin	Homo sapiens	50-58	
1534272-9	1992	An RA-resistant clone, designated HL-60/R3, constitutively expressed larger amounts of alpha-actinin, vinculin, lipocortin I, and lipocortin II than parental HL-60 cells.	Tretinoin	3-5	vinculin	Homo sapiens	102-110