PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 23587524-4 2013 ATRA induced upregulation of Pyk2 mRNA, protein and phosphorylation levels and enhanced Pyk2 interaction with paxillin and vinculin. Tretinoin 0-4 vinculin Homo sapiens 123-131 6188622-0 1983 Formation of vinculin plaques precedes other cytoskeletal changes during retinoic acid-induced teratocarcinoma cell differentiation. Tretinoin 73-86 vinculin Homo sapiens 13-21 6188622-8 1983 This sequence of changes suggests that the vinculin-containing adhesion plaques may be important in the mechanism of RA-induced differentiation of EC cells. Tretinoin 117-119 vinculin Homo sapiens 43-51 3099098-7 1986 The changes in PK-C activity in TPA + RA-treated cells were accompanied by Ca2+/phospholipid(PL)-dependent phosphorylation in vitro of pp38 which is characteristic of treatment with RA alone, as well as the Ca2+/PL-independent phosphorylation in vitro of pp82 and pp130 (vinculin) which is prevalent in cells treated continuously with TPA alone and is absent in RA-treated cells. Tretinoin 38-40 vinculin Homo sapiens 271-279 32254654-11 2018 Both cell types exhibited more vinculin expression when seeded to RA-loaded PCL scaffolds. Tretinoin 66-68 vinculin Homo sapiens 31-39 1534272-6 1992 In contrast, cells exposed to RA contained larger amounts of alpha-actinin, vinculin, talin, lipocortin I, and lipocortin II, as determined with their respective antibodies followed by flow cytometric analysis as described above. Tretinoin 30-32 vinculin Homo sapiens 76-84 1534272-8 1992 Treatment of HL-60/S4 with RA led to increases in vinculin, talin, lipocortin I, and lipocortin II. Tretinoin 27-29 vinculin Homo sapiens 50-58 1534272-9 1992 An RA-resistant clone, designated HL-60/R3, constitutively expressed larger amounts of alpha-actinin, vinculin, lipocortin I, and lipocortin II than parental HL-60 cells. Tretinoin 3-5 vinculin Homo sapiens 102-110