PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25972084-2	2015	Restin was firstly cloned from HL-60 cells treated with all-trans retinoic acid (ATRA).	Tretinoin	56-79	MAGE family member H1	Homo sapiens	0-6	
25972084-2	2015	Restin was firstly cloned from HL-60 cells treated with all-trans retinoic acid (ATRA).	Tretinoin	81-85	MAGE family member H1	Homo sapiens	0-6	
17393088-1	2007	Restin, belonging to the melanoma-associated antigen superfamily, was firstly cloned from the differentiated HL-60 cells when induced by all-trans retinoic acid (ATRA) in our lab.	Tretinoin	147-160	MAGE family member H1	Homo sapiens	0-6	
17393088-1	2007	Restin, belonging to the melanoma-associated antigen superfamily, was firstly cloned from the differentiated HL-60 cells when induced by all-trans retinoic acid (ATRA) in our lab.	Tretinoin	162-166	MAGE family member H1	Homo sapiens	0-6	
18759029-1	2002	In the present study, a new member of melanoma associated antigens (Mage), named Restin (219 amino acids), was identified from HL-60 cell induced by all-trans-retinoic acid (ATRA) by PCR-based subtractive hybridization.	Tretinoin	149-172	MAGE family member H1	Homo sapiens	81-87	
18759029-1	2002	In the present study, a new member of melanoma associated antigens (Mage), named Restin (219 amino acids), was identified from HL-60 cell induced by all-trans-retinoic acid (ATRA) by PCR-based subtractive hybridization.	Tretinoin	174-178	MAGE family member H1	Homo sapiens	81-87	
16574066-1	2006	RESTIN, a member of the melanoma-associated antigen superfamily, is a nuclear protein induced by atRA (all-trans retinoic acid) in HL60 cells.	Tretinoin	97-101	MAGE family member H1	Homo sapiens	0-6	
16574066-1	2006	RESTIN, a member of the melanoma-associated antigen superfamily, is a nuclear protein induced by atRA (all-trans retinoic acid) in HL60 cells.	Tretinoin	103-126	MAGE family member H1	Homo sapiens	0-6	
16574066-4	2006	In this study, we observed that up-regulation of restin was present during the atRA-induced HL60 cell differentiation process, suggesting the functional relevance between RESTIN and atRA-induced cellular effects.	Tretinoin	79-83	MAGE family member H1	Homo sapiens	49-55	
16574066-4	2006	In this study, we observed that up-regulation of restin was present during the atRA-induced HL60 cell differentiation process, suggesting the functional relevance between RESTIN and atRA-induced cellular effects.	Tretinoin	79-83	MAGE family member H1	Homo sapiens	171-177	
16574066-4	2006	In this study, we observed that up-regulation of restin was present during the atRA-induced HL60 cell differentiation process, suggesting the functional relevance between RESTIN and atRA-induced cellular effects.	Tretinoin	182-186	MAGE family member H1	Homo sapiens	49-55	
16574066-7	2006	Interestingly, restin promoter containing several potential consensus-binding sites of STAT-1alpha was activated by atRA in ER(+) MCF-7 cells but not in ER(-) MDA-MB-231 cells, over-expression of STAT-1alpha in latter rescued the activation effect of restin promoter in response to atRA and IFNgamma.	Tretinoin	116-120	MAGE family member H1	Homo sapiens	4-10	
16574066-7	2006	Interestingly, restin promoter containing several potential consensus-binding sites of STAT-1alpha was activated by atRA in ER(+) MCF-7 cells but not in ER(-) MDA-MB-231 cells, over-expression of STAT-1alpha in latter rescued the activation effect of restin promoter in response to atRA and IFNgamma.	Tretinoin	116-120	MAGE family member H1	Homo sapiens	15-21	
16574066-8	2006	Our evidence supported that STAT-1alpha plays an important role in the atRA-induced transcriptional up-regulation of restin, which was associated with the atRA-induced HL60 cell differentiation and potentially mediated the downstream effects of atRA signal pathway via STAT-1alpha in some cancer cells.	Tretinoin	71-75	MAGE family member H1	Homo sapiens	117-123	
16092758-1	2005	Restin, a member of melanoma-associated antigen superfamily gene, was first cloned from differentiated leukemia cell induced by all trans-retinoic acid, and was able to inhibit cell proliferation, but the molecular mechanism was not clear.	Tretinoin	132-151	MAGE family member H1	Homo sapiens	0-6	
15694019-1	2005	BACKGROUND & OBJECTIVE: apr-1 was cloned by improved polymerase chain reaction (PCR)-based subtractive hybridization from all-trans retinoic acid (ATRA)-induced apoptotic leukemia HL-60 cells in 1999.	Tretinoin	136-149	MAGE family member H1	Homo sapiens	28-33	
15694019-1	2005	BACKGROUND & OBJECTIVE: apr-1 was cloned by improved polymerase chain reaction (PCR)-based subtractive hybridization from all-trans retinoic acid (ATRA)-induced apoptotic leukemia HL-60 cells in 1999.	Tretinoin	151-155	MAGE family member H1	Homo sapiens	28-33