PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10753864-13	2000	Furthermore, RA treatment enhanced the transcriptional activity of a reporter construct containing the Sry/Sox consensus sequence in TC6 cells.	Tretinoin	13-15	quiescin Q6 sulfhydryl oxidase 1	Mus musculus	107-110	
11278854-9	2001	We found that mutation of the SOX/OCT site and targeted inactivation of Hoxa1 both impair the response of the b1-ARE to retinoic acid in transgenic mice.	Tretinoin	120-133	quiescin Q6 sulfhydryl oxidase 1	Mus musculus	30-33	
11278854-10	2001	Our results show that Hoxa1 is the primary mediator of the response of b1-ARE to retinoic acid in vivo and that this function is dependent on the binding of SOX/OCT heterodimers to the b1-ARE.	Tretinoin	81-94	quiescin Q6 sulfhydryl oxidase 1	Mus musculus	157-160	
23007396-10	2012	We show specific effects on the Wnt-catenin pathway and on members of the Runx, Fox, and Sox transcription factor families, indicating that RA modulates pathways and factors implicated in hf downgrowth and bending.	Tretinoin	140-142	quiescin Q6 sulfhydryl oxidase 1	Mus musculus	89-92