PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10366427-0	1999	Role of vitamin D3 receptor in the synergistic differentiation of WEHI-3B leukemia cells by vitamin D3 and retinoic acid.	Tretinoin	107-120	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	8-27	
26504088-1	2015	The biological actions of 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) are mediated by the vitamin D receptor (VDR), whose expression in bone cells is regulated positively by 1,25(OH)2D3, retinoic acid, and parathyroid hormone through both intergenic and intronic enhancers.	Tretinoin	182-195	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	85-103	
26504088-1	2015	The biological actions of 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) are mediated by the vitamin D receptor (VDR), whose expression in bone cells is regulated positively by 1,25(OH)2D3, retinoic acid, and parathyroid hormone through both intergenic and intronic enhancers.	Tretinoin	182-195	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	105-108	
19474283-11	2009	CONCLUSIONS: These results suggested selective cooperation of RAR and VDR in the regulation of the nephrin gene, i.e. (1) ATRA induces nephrin gene expression via RAR independently of RXR and VDR and (2) 1,25(OH)(2)D(3) induces nephrin gene expression via selective cooperation of RAR and VDR, which is independent of RXR.	Tretinoin	122-126	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	70-73	
14666262-5	2003	The possibility that retinoic acid would induce the expression of the vitamin D receptor and synergize with vitamin D, a known inhibitor of HC11 cell growth, was also investigated.	Tretinoin	21-34	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	70-88	
28635660-0	2017	Diverse Regulation of Vitamin D Receptor Gene Expression by 1,25-Dihydroxyvitamin D and ATRA in Murine and Human Blood Cells at Early Stages of Their Differentiation.	Tretinoin	88-92	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	22-40	
10366427-4	1999	No VDR was detected in untreated WEHI-3B D- cells; however, RA and 1,25-(OH)2D3 when used as single agents caused a slight induction of the VDR and in combination produced a marked increase in the VDR.	Tretinoin	60-62	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	140-143	
10366427-4	1999	No VDR was detected in untreated WEHI-3B D- cells; however, RA and 1,25-(OH)2D3 when used as single agents caused a slight induction of the VDR and in combination produced a marked increase in the VDR.	Tretinoin	60-62	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	140-143	
10366427-9	1999	These findings suggest that (a) induction of VDR expression is a key component in the synergistic differentiation induced by 1,25-(OH)2D3 and RA and (b) RAR and not RXR must be activated for enhanced induction of the VDR and for the synergistic differentiation produced by RA and 1, 25-(OH)2D3.	Tretinoin	142-144	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	45-48	
10366427-9	1999	These findings suggest that (a) induction of VDR expression is a key component in the synergistic differentiation induced by 1,25-(OH)2D3 and RA and (b) RAR and not RXR must be activated for enhanced induction of the VDR and for the synergistic differentiation produced by RA and 1, 25-(OH)2D3.	Tretinoin	153-155	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	45-48	
10366427-9	1999	These findings suggest that (a) induction of VDR expression is a key component in the synergistic differentiation induced by 1,25-(OH)2D3 and RA and (b) RAR and not RXR must be activated for enhanced induction of the VDR and for the synergistic differentiation produced by RA and 1, 25-(OH)2D3.	Tretinoin	153-155	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	217-220	
8574944-1	1995	The effects of retinoic acid (RA), and calcitriol are mediated by specific nuclear receptors (RARs and VDR, respectively).	Tretinoin	15-28	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	103-106	
8574944-1	1995	The effects of retinoic acid (RA), and calcitriol are mediated by specific nuclear receptors (RARs and VDR, respectively).	Tretinoin	30-32	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	103-106	
8382933-6	1993	In contrast to the effects of RA on osteoblast-related gene regulation, RA was found to increase the quantity of estrogen receptor as well as of 1,25-dihydroxy vitamin D3 receptor (VDR) in BFO cells.	Tretinoin	72-74	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	160-179	
8203489-6	1994	Although RA increased VDR levels, RA did not potentiate the effect of 1,25(OH)2D on ALP activity.	Tretinoin	9-11	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	22-25	
8382933-6	1993	In contrast to the effects of RA on osteoblast-related gene regulation, RA was found to increase the quantity of estrogen receptor as well as of 1,25-dihydroxy vitamin D3 receptor (VDR) in BFO cells.	Tretinoin	72-74	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	181-184	
8382933-8	1993	These RA effects on ER and VDR seem to be specific, since glucocorticoid receptor quantities were not affected by RA treatment.	Tretinoin	6-8	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	27-30	
32866959-5	2021	In summary, we conclude that epidermal hyperproliferation of mouse skin in response to a topically administered vitamin D receptor antagonist/partial agonist (ZK159222) is induced via increased retinoic acid synthesis, retinoic acid levels and increased RARgamma-mediated pathways.	Tretinoin	194-207	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	112-130	
32866959-5	2021	In summary, we conclude that epidermal hyperproliferation of mouse skin in response to a topically administered vitamin D receptor antagonist/partial agonist (ZK159222) is induced via increased retinoic acid synthesis, retinoic acid levels and increased RARgamma-mediated pathways.	Tretinoin	219-232	vitamin D (1,25-dihydroxyvitamin D3) receptor	Mus musculus	112-130