PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 23909735-0 2013 Vitamin D transport proteins megalin and disabled-2 are expressed in prostate and colon epithelial cells and are induced and activated by all-trans-retinoic acid. Tretinoin 138-161 DAB adaptor protein 2 Homo sapiens 41-51 11577091-10 2001 Thus, Dab2 is one of the principal genes induced by retinoic acid involved in cell growth suppression, and expression of Dab2 alone is sufficient for uncoupling of MAPK activation and c-Fos expression. Tretinoin 52-65 DAB adaptor protein 2 Homo sapiens 6-10 11577091-11 2001 Resistance to retinoic acid regulation in PA-1 cells likely results from defects in retinoic acid up-regulation of Dab2 expression. Tretinoin 14-27 DAB adaptor protein 2 Homo sapiens 115-119 23909735-7 2013 Megalin and Dab2 were expressed in prostate and colon epithelial cells, which was markedly enhanced following treatment with RA. Tretinoin 125-127 DAB adaptor protein 2 Homo sapiens 12-16 23909735-4 2013 Moreover, induction of megalin and Dab2 (protein and mRNA abundance) by all-trans-retinoic acid (RA) enhanced DBP uptake. Tretinoin 72-95 DAB adaptor protein 2 Homo sapiens 35-39 23909735-4 2013 Moreover, induction of megalin and Dab2 (protein and mRNA abundance) by all-trans-retinoic acid (RA) enhanced DBP uptake. Tretinoin 97-99 DAB adaptor protein 2 Homo sapiens 35-39 22525672-1 2012 F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2). Tretinoin 106-119 DAB adaptor protein 2 Homo sapiens 219-229 22525672-1 2012 F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2). Tretinoin 106-119 DAB adaptor protein 2 Homo sapiens 231-235 22525672-1 2012 F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2). Tretinoin 121-123 DAB adaptor protein 2 Homo sapiens 219-229 22525672-1 2012 F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2). Tretinoin 121-123 DAB adaptor protein 2 Homo sapiens 231-235 22525672-3 2012 During the RA-induced differentiation process, Dab2 expression is induced by the GATA factors in a coherent feed-forward loop; on the other hand, we showed that p96 regulates GATA-4 in a positive feed-back manner in this study. Tretinoin 11-13 DAB adaptor protein 2 Homo sapiens 47-51 18567755-5 2008 Using immunoblotting and real-time PCR, we found that megalin, cubilin, and Dab2 were expressed and dose dependently induced by all-trans-retinoic acid (RA) in T-47D human breast cancer cells and that RA-treated T-47D cells exhibited enhanced DBP internalization. Tretinoin 132-151 DAB adaptor protein 2 Homo sapiens 76-80 18567755-5 2008 Using immunoblotting and real-time PCR, we found that megalin, cubilin, and Dab2 were expressed and dose dependently induced by all-trans-retinoic acid (RA) in T-47D human breast cancer cells and that RA-treated T-47D cells exhibited enhanced DBP internalization. Tretinoin 153-155 DAB adaptor protein 2 Homo sapiens 76-80 18567755-5 2008 Using immunoblotting and real-time PCR, we found that megalin, cubilin, and Dab2 were expressed and dose dependently induced by all-trans-retinoic acid (RA) in T-47D human breast cancer cells and that RA-treated T-47D cells exhibited enhanced DBP internalization. Tretinoin 201-203 DAB adaptor protein 2 Homo sapiens 76-80