PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28203527-12	2016	Angiotensin II increased PKC activity in vehicle-treated cells that further increased in DETC-treated cells, which was attenuated by AT1R blocker candesartan and SOD-mimetic tempol.	Ditiocarb	89-93	angiotensinogen	Homo sapiens	0-14	
18756259-1	2008	BACKGROUND: The aim of this study was to determine whether inhibition of superoxide dismutase (SOD) with diethyldithiocarbamic acid (DETC) could affect the collagen production, the mRNA and protein expression of collagen types I and III, and fibronectin in control and angiotensin II (ANG II)-treated cardiac fibroblasts.	Ditiocarb	105-131	angiotensinogen	Homo sapiens	269-283	
18756259-1	2008	BACKGROUND: The aim of this study was to determine whether inhibition of superoxide dismutase (SOD) with diethyldithiocarbamic acid (DETC) could affect the collagen production, the mRNA and protein expression of collagen types I and III, and fibronectin in control and angiotensin II (ANG II)-treated cardiac fibroblasts.	Ditiocarb	105-131	angiotensinogen	Homo sapiens	285-291	
18756259-1	2008	BACKGROUND: The aim of this study was to determine whether inhibition of superoxide dismutase (SOD) with diethyldithiocarbamic acid (DETC) could affect the collagen production, the mRNA and protein expression of collagen types I and III, and fibronectin in control and angiotensin II (ANG II)-treated cardiac fibroblasts.	Ditiocarb	133-137	angiotensinogen	Homo sapiens	269-283	
18756259-1	2008	BACKGROUND: The aim of this study was to determine whether inhibition of superoxide dismutase (SOD) with diethyldithiocarbamic acid (DETC) could affect the collagen production, the mRNA and protein expression of collagen types I and III, and fibronectin in control and angiotensin II (ANG II)-treated cardiac fibroblasts.	Ditiocarb	133-137	angiotensinogen	Homo sapiens	285-291	
18756259-4	2008	Superoxide anion production was increased by DETC and decreased by tempol in control and ANG II-treated fibroblasts.	Ditiocarb	45-49	angiotensinogen	Homo sapiens	89-95	
18756259-5	2008	DETC also reduced the intracellular generation of reactive oxygen species (ROS) (such as H2O2, hydroxyl radicals, hydroperoxides) in control and ANG II-treated fibroblasts, whereas tempol reduced the ROS production only in ANG II-treated fibroblasts.	Ditiocarb	0-4	angiotensinogen	Homo sapiens	145-151	
18756259-5	2008	DETC also reduced the intracellular generation of reactive oxygen species (ROS) (such as H2O2, hydroxyl radicals, hydroperoxides) in control and ANG II-treated fibroblasts, whereas tempol reduced the ROS production only in ANG II-treated fibroblasts.	Ditiocarb	0-4	angiotensinogen	Homo sapiens	223-229	
12573135-3	2002	Ang II induced a quick and transient increase of dichlorodihydrofluorescein (DCHF) fluorescence in VSMC, an effect that was completely abolished by catalase and by diethyldithiocarbamate, a cell-permeable superoxide dismutase inhibitor.	Ditiocarb	164-186	angiotensinogen	Homo sapiens	0-6