PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10452962-6	1999	H(2)O(2) addition to i-NOS macrophages also caused cell death which was, in part, prevented by DETC or urate.	Ditiocarb	95-99	nitric oxide synthase 2	Homo sapiens	21-26	
9545547-6	1998	Administration of diethyldithiocarbamate (DDC) inhibited induction of NF-kappa B and concomitantly the expression of IL-1 beta, IL-6, TNF-alpha as well as iNOS.	Ditiocarb	18-40	nitric oxide synthase 2	Homo sapiens	155-159	
9545547-6	1998	Administration of diethyldithiocarbamate (DDC) inhibited induction of NF-kappa B and concomitantly the expression of IL-1 beta, IL-6, TNF-alpha as well as iNOS.	Ditiocarb	42-45	nitric oxide synthase 2	Homo sapiens	155-159	
15715934-0	2005	Diethyldithiocarbamate inhibits iNOS expression in human lens epithelial cells stimulated by IFN-gamma and LPS.	Ditiocarb	0-22	nitric oxide synthase 2	Homo sapiens	32-36	
15715934-5	2005	The expression of iNOS was significantly reduced in a dose-dependent manner by adding DDC from 10 micromol/L to 1 mmol/L.	Ditiocarb	86-89	nitric oxide synthase 2	Homo sapiens	18-22	
15715934-6	2005	CONCLUSION: The expression of iNOS in HLEC needs co-stimulation with IFN-gamma and LPS and it is inhibited by DDC.	Ditiocarb	110-113	nitric oxide synthase 2	Homo sapiens	30-34	
15482639-6	2004	Semiquantitative reverse transcription polymerase chain reaction (RT-PCR) was performed to study the effect of diethyldithiocarbamate (DDC), a metabolite of Zn-DDC, on the transcription inducible nitric oxide synthase (iNOS) mRNA in human lens epithelial cells (HLEC).	Ditiocarb	111-133	nitric oxide synthase 2	Homo sapiens	186-217	
15482639-6	2004	Semiquantitative reverse transcription polymerase chain reaction (RT-PCR) was performed to study the effect of diethyldithiocarbamate (DDC), a metabolite of Zn-DDC, on the transcription inducible nitric oxide synthase (iNOS) mRNA in human lens epithelial cells (HLEC).	Ditiocarb	111-133	nitric oxide synthase 2	Homo sapiens	219-223	
15482639-6	2004	Semiquantitative reverse transcription polymerase chain reaction (RT-PCR) was performed to study the effect of diethyldithiocarbamate (DDC), a metabolite of Zn-DDC, on the transcription inducible nitric oxide synthase (iNOS) mRNA in human lens epithelial cells (HLEC).	Ditiocarb	135-138	nitric oxide synthase 2	Homo sapiens	186-217	
15482639-6	2004	Semiquantitative reverse transcription polymerase chain reaction (RT-PCR) was performed to study the effect of diethyldithiocarbamate (DDC), a metabolite of Zn-DDC, on the transcription inducible nitric oxide synthase (iNOS) mRNA in human lens epithelial cells (HLEC).	Ditiocarb	135-138	nitric oxide synthase 2	Homo sapiens	219-223	
15482639-6	2004	Semiquantitative reverse transcription polymerase chain reaction (RT-PCR) was performed to study the effect of diethyldithiocarbamate (DDC), a metabolite of Zn-DDC, on the transcription inducible nitric oxide synthase (iNOS) mRNA in human lens epithelial cells (HLEC).	Ditiocarb	157-163	nitric oxide synthase 2	Homo sapiens	186-217	
15482639-14	2004	DDC, the metabolite of Zn-DDC, may be one of the factors in the prevention of cataract formation because it inhibits the transcription of iNOS mRNA.	Ditiocarb	0-3	nitric oxide synthase 2	Homo sapiens	138-142	
15482639-14	2004	DDC, the metabolite of Zn-DDC, may be one of the factors in the prevention of cataract formation because it inhibits the transcription of iNOS mRNA.	Ditiocarb	23-29	nitric oxide synthase 2	Homo sapiens	138-142