PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30866411-7	2019	Thus, reactive oxygen species appear as a highly active proapoptotic pathway in CTCL, which may be promising for therapeutic intervention.	Reactive Oxygen Species	6-29	TSPY like 2	Homo sapiens	80-84	
28729399-6	2017	Present studies demonstrate that exposure of CTCL cells to decitabine in combination with GO-203, increased the generation of reactive oxygen species (ROS) levels and decreased levels of scavenger molecules, NADP, NADPH, glutathione, and TIGAR, critical to intracellular redox homeostasis.	Reactive Oxygen Species	126-149	TSPY like 2	Homo sapiens	45-49	
28729399-6	2017	Present studies demonstrate that exposure of CTCL cells to decitabine in combination with GO-203, increased the generation of reactive oxygen species (ROS) levels and decreased levels of scavenger molecules, NADP, NADPH, glutathione, and TIGAR, critical to intracellular redox homeostasis.	Reactive Oxygen Species	151-154	TSPY like 2	Homo sapiens	45-49	
26048911-3	2015	Although CTCL cells exhibit resistance to ROS-induced apoptosis, the expression and functional significance of MUC1 in CTCL have not been previously investigated.	Reactive Oxygen Species	42-45	TSPY like 2	Homo sapiens	9-13	
26048911-7	2015	In concert with these results, targeting MUC1-C in CTCL cells increased ROS and, in turn, induced ROS-mediated late apoptosis/necrosis.	Reactive Oxygen Species	72-75	TSPY like 2	Homo sapiens	51-55	
26048911-7	2015	In concert with these results, targeting MUC1-C in CTCL cells increased ROS and, in turn, induced ROS-mediated late apoptosis/necrosis.	Reactive Oxygen Species	98-101	TSPY like 2	Homo sapiens	51-55