PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33203990-5	2021	Mechanistically, AMD1 depletion induced loss of mitochondrial membrane potential and accumulation of reactive oxygen species (ROS), resulting in the differentiation of LSCs via oxidative stress and aberrant activation of unfolded protein response (UPR) pathway, which was partially rescued by the addition of polyamine.	Reactive Oxygen Species	101-124	adenosylmethionine decarboxylase 1	Homo sapiens	17-21	
17069747-0	2006	S-Adenosylmethionine decarboxylase partially regulates cell growth of HL-60 cells by controlling the intracellular ROS level: Early senescence and sensitization to gamma-radiation.	Reactive Oxygen Species	115-118	adenosylmethionine decarboxylase 1	Homo sapiens	0-34	
17069747-2	2006	SAMDC-suppressed HL-60 cells overproduced intracellular reactive oxygen species (ROS), which led to cell growth defect and partial cell death.	Reactive Oxygen Species	56-79	adenosylmethionine decarboxylase 1	Homo sapiens	0-5	
17069747-2	2006	SAMDC-suppressed HL-60 cells overproduced intracellular reactive oxygen species (ROS), which led to cell growth defect and partial cell death.	Reactive Oxygen Species	81-84	adenosylmethionine decarboxylase 1	Homo sapiens	0-5	
33203990-5	2021	Mechanistically, AMD1 depletion induced loss of mitochondrial membrane potential and accumulation of reactive oxygen species (ROS), resulting in the differentiation of LSCs via oxidative stress and aberrant activation of unfolded protein response (UPR) pathway, which was partially rescued by the addition of polyamine.	Reactive Oxygen Species	126-129	adenosylmethionine decarboxylase 1	Homo sapiens	17-21