PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22041456-8	2012	These results support the hypothesis that mutations in SDHD may contribute to carcinogenesis by increasing genomic instability mediated by increased steady-state levels of reactive oxygen species.	Reactive Oxygen Species	172-195	succinate dehydrogenase complex subunit D	Homo sapiens	55-59	
25328978-0	2015	Instability of succinate dehydrogenase in SDHD polymorphism connects reactive oxygen species production to nuclear and mitochondrial genomic mutations in yeast.	Reactive Oxygen Species	69-92	succinate dehydrogenase complex subunit D	Homo sapiens	42-46	
25328978-10	2015	INNOVATION AND CONCLUSION: SDHD mutations are associated with protein and nuclear and mitochondrial genomic instability and increase ROS production in our yeast model.	Reactive Oxygen Species	133-136	succinate dehydrogenase complex subunit D	Homo sapiens	27-31