PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24724551-6	2014	Light irradiation stimulated ROS-dependent actin polymerization, and this effect was abrogated by PBP10, a peptide which activates the actin-severing protein, gelsolin, and causes actin-depolymerization in human sperm.	Reactive Oxygen Species	29-32	gelsolin	Homo sapiens	159-167	
27391159-0	2016	Gelsolin-Cu/ZnSOD interaction alters intracellular reactive oxygen species levels to promote cancer cell invasion.	Reactive Oxygen Species	51-74	gelsolin	Homo sapiens	0-8	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	3-26	gelsolin	Homo sapiens	121-129	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	3-26	gelsolin	Homo sapiens	247-255	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	3-26	gelsolin	Homo sapiens	247-255	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	28-31	gelsolin	Homo sapiens	121-129	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	28-31	gelsolin	Homo sapiens	247-255	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	28-31	gelsolin	Homo sapiens	247-255	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	149-152	gelsolin	Homo sapiens	121-129	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	149-152	gelsolin	Homo sapiens	247-255	
27391159-3	2016	As reactive oxygen species (ROS) have been shown to promote cancer cell invasion, we investigated on the hypothesis that gelsolin-induced changes in ROS levels may mediate the invasive capacity of colon cancer cells.Herein, we show that increased gelsolin enhances the invasive capacity of colon cancer cells, and this is mediated via gelsolin"s effects in elevating intracellular superoxide (O2.-) levels.	Reactive Oxygen Species	149-152	gelsolin	Homo sapiens	247-255	
15625007-5	2004	RESULTS: We report that a cell permeable ten amino acid peptide (PBP10) derived from the phosphatidylinositol 4,5-bisphosphate (PIP2) binding region of gelsolin (an uncapper of actin filaments) blocks granule mobilization as well as secretion of oxygen radicals.	Reactive Oxygen Species	246-261	gelsolin	Homo sapiens	152-160