PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34315867-11	2021	In addition, we confirmed that the opposite effects on ferroptosis and ferroptosis-associated lipid reactive oxygen species (ROS) generation resulted from knockout and overexpression of TRIB2 were all indispensible of TFRC and betaTrCP.	Reactive Oxygen Species	125-128	beta-transducin repeat containing E3 ubiquitin protein ligase	Homo sapiens	227-235	
26935904-12	2016	However, VEGFR-2 was reduced when directly exposed to ROS, with resultant co-location of beta-TrCP and VEGFR-2.	Reactive Oxygen Species	54-57	beta-transducin repeat containing E3 ubiquitin protein ligase	Homo sapiens	89-98	
26935904-14	2016	Decreased VEGFR-2 caused by ROS was ameliorated by beta-TrCP siRNA, proteasome inhibitor MG132 and GSK-3beta activity inhibitor (lithium chloride and SB216763).	Reactive Oxygen Species	28-31	beta-transducin repeat containing E3 ubiquitin protein ligase	Homo sapiens	51-60	
26935904-15	2016	We suggest that redundant ROS reduces VEGFR-2 through beta-TrCP-mediated VEGFR-2 degradation, which is postulated to be regulated by GSK-3beta.	Reactive Oxygen Species	26-29	beta-transducin repeat containing E3 ubiquitin protein ligase	Homo sapiens	54-63