PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20349048-9	2010	Finally, it was observed that the nitric oxide (NO) anti-inflammatory effects on ICAM-1 expression appear to be indirectly mediated by HO-1 activation, since the inhibition of HO-1 prevented the inhibitory effect of the NO donor (S-nitroso-N-acetylpenicillamine) on LPS-induced ICAM-1 expression.	S-Nitroso-N-Acetylpenicillamine	230-261	heme oxygenase 1	Homo sapiens	135-139	
20349048-9	2010	Finally, it was observed that the nitric oxide (NO) anti-inflammatory effects on ICAM-1 expression appear to be indirectly mediated by HO-1 activation, since the inhibition of HO-1 prevented the inhibitory effect of the NO donor (S-nitroso-N-acetylpenicillamine) on LPS-induced ICAM-1 expression.	S-Nitroso-N-Acetylpenicillamine	230-261	heme oxygenase 1	Homo sapiens	176-180	
10780998-5	2000	Incubation of cells with NO donors, spermine nonoate (SPNO) and S-nitroso-N-acetylpenicillamine (SNAP), induced a dose- and time-dependent expression of HO-1 protein.	S-Nitroso-N-Acetylpenicillamine	64-95	heme oxygenase 1	Homo sapiens	153-157	
10780998-5	2000	Incubation of cells with NO donors, spermine nonoate (SPNO) and S-nitroso-N-acetylpenicillamine (SNAP), induced a dose- and time-dependent expression of HO-1 protein.	S-Nitroso-N-Acetylpenicillamine	97-101	heme oxygenase 1	Homo sapiens	153-157