PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20349048-9	2010	Finally, it was observed that the nitric oxide (NO) anti-inflammatory effects on ICAM-1 expression appear to be indirectly mediated by HO-1 activation, since the inhibition of HO-1 prevented the inhibitory effect of the NO donor (S-nitroso-N-acetylpenicillamine) on LPS-induced ICAM-1 expression.	S-Nitroso-N-Acetylpenicillamine	230-261	intercellular adhesion molecule 1	Homo sapiens	81-87	
20349048-9	2010	Finally, it was observed that the nitric oxide (NO) anti-inflammatory effects on ICAM-1 expression appear to be indirectly mediated by HO-1 activation, since the inhibition of HO-1 prevented the inhibitory effect of the NO donor (S-nitroso-N-acetylpenicillamine) on LPS-induced ICAM-1 expression.	S-Nitroso-N-Acetylpenicillamine	230-261	intercellular adhesion molecule 1	Homo sapiens	278-284	
9344755-10	1997	Finally, an exogenous NO donor, S-nitrosoacetyl-penicillamine (SNAP, 100 microM), suppressed the generation of ROS upon reoxygenation, and blocked the activation of NFkappaB and the upregulation of ICAM-1.	S-Nitroso-N-Acetylpenicillamine	32-61	intercellular adhesion molecule 1	Homo sapiens	198-204	
9344755-10	1997	Finally, an exogenous NO donor, S-nitrosoacetyl-penicillamine (SNAP, 100 microM), suppressed the generation of ROS upon reoxygenation, and blocked the activation of NFkappaB and the upregulation of ICAM-1.	S-Nitroso-N-Acetylpenicillamine	63-67	intercellular adhesion molecule 1	Homo sapiens	198-204