PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15252149-11	2004	In summary, selenite-induced p53 Ser15P appeared to be important for activating the caspase-mediated apoptosis involving both the caspase-8 and the caspase-9 pathways in the LNCaP cells.	Selenious Acid	12-20	caspase 8	Homo sapiens	130-139	
24577083-7	2014	Collectively, our findings demonstrate that selenite caused CYLD upregulation via LEF1 and cIAP downregulation, both of which contribute to the degradation of ubiquitin chains on RIP1 and subsequent caspase-8 activation and apoptosis.	Selenious Acid	44-52	caspase 8	Homo sapiens	199-208	
19189638-6	2008	Conversely, RKO cells expressing wildtype p53, proteolytically activated caspase-8, -9, -7 and -3 and unmethylated caspase-8 were more responsive to 5-FU and selenous acid-induced apoptosis.	Selenious Acid	158-171	caspase 8	Homo sapiens	73-97	
19189638-6	2008	Conversely, RKO cells expressing wildtype p53, proteolytically activated caspase-8, -9, -7 and -3 and unmethylated caspase-8 were more responsive to 5-FU and selenous acid-induced apoptosis.	Selenious Acid	158-171	caspase 8	Homo sapiens	73-82	
23589036-13	2013	Western blot analyses showed that selenite-induced apoptosis was accompanied by activation of caspase-8 and specific proteolytic cleavage of PARP.	Selenious Acid	34-42	caspase 8	Homo sapiens	94-103