PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31836396-12	2020	DISCUSSION: Homozygosity for the common ACADM p.Lys329Glu pathogenic variant was associated with increased levels of C8-carnitine and transaminases.	Carnitine	117-129	acyl-CoA dehydrogenase medium chain	Homo sapiens	40-45	
22630369-9	2012	After diagnosis and initiation of treatment, residual MCAD enzyme activities <10% were associated with an increased risk of hypoglycaemia and carnitine supplementation.	Carnitine	145-154	acyl-CoA dehydrogenase medium chain	Homo sapiens	54-58	
19080238-7	2008	CONCLUSION: Metabolic disorder and cardiac remodeling occur in the development process of ACM; they are partly prevented by L-carnitine through downregulating mRNA and protein expressions of PPARalpha, RXRalpha, CPT-I, MCAD and PPARgamma.	Carnitine	124-135	acyl-CoA dehydrogenase medium chain	Homo sapiens	219-223