PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15726889-6	2004	Studies were also conducted on the effect of the two fucoidans and heparin on the activation of Glu-Plg by t-PA using 0.05M Tris buffer pH 7.4 containing 0.1 M NaCl.	Glutamic Acid	96-99	chromosome 20 open reading frame 181	Homo sapiens	107-111	
22435890-8	2012	Treatment with this tPA variant provides a novel approach for limiting neuronal toxicity caused by untoward NMDA-receptor activation mediated by increased tPA and glutamate following TBI.	Glutamic Acid	163-172	chromosome 20 open reading frame 181	Homo sapiens	20-23	
15496678-4	2004	Here, we show tPA activity is rapidly increased in hippocampal neurons after glutamate stimulation.	Glutamic Acid	77-86	chromosome 20 open reading frame 181	Homo sapiens	14-17	
15496678-6	2004	Furthermore, the mRNA encoding tPA is present in dendrites and is rapidly polyadenylated after glutamate stimulation.	Glutamic Acid	95-104	chromosome 20 open reading frame 181	Homo sapiens	31-34	
14743974-7	2003	However, upon increasing the NaCl to 0.9%, the results showed that the enhancements by 6-AH of the activation of Glu-Plg by streptokinase or t-PA were related to its interaction with Glu-Plg.	Glutamic Acid	113-116	chromosome 20 open reading frame 181	Homo sapiens	141-145	
3129815-4	1988	On the other hand, UK and t-PA rapidly activated Glu-plg in the clotted plasma.	Glutamic Acid	49-52	chromosome 20 open reading frame 181	Homo sapiens	26-30	
3127307-1	1988	The activation of Glu1-plasminogen (Glu-Pg) by streptokinase (SK), urokinase (UK) and tissue plasminogen activator (tPA) is under rigorous control by molecules such as epsilon-aminocaproic acid (EACA), fibrinogen (Fg), fibrin (Fn) and, as we have recently discovered, anions.	Glutamic Acid	18-21	chromosome 20 open reading frame 181	Homo sapiens	86-114