PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9918228-5	1999	Glutamate or the mixture of agonists also induced a 56 kDa band identified as Fos protein in SCN tissue.	Glutamic Acid	0-9	proto-oncogene c-Fos	Mesocricetus auratus	78-81	
9918228-6	1999	In dissociated cultures of SCN, glutamate caused a rapid (15 min) induction of nuclear pCREB-ir and Fos-ir (after 60 min) exclusively in neurones, both GABA-ir and others.	Glutamic Acid	32-41	proto-oncogene c-Fos	Mesocricetus auratus	100-103	
9918228-10	1999	The effects of glutamate on pCREB-ir and Fos-ir were blocked by antagonists of both NMDA (MK801) and AMPA/KA (NBQX) receptors.	Glutamic Acid	15-24	proto-oncogene c-Fos	Mesocricetus auratus	41-44	
9918228-12	1999	However, the AMPA/KA receptor antagonist was no longer effective at blocking glutamatergic induction of either Fos-ir or pCREB-ir, consistent with the model that glutamate regulates gene expression in the SCN by a co-ordinate action through both NMDA and AMPA/KA receptors.	Glutamic Acid	77-86	proto-oncogene c-Fos	Mesocricetus auratus	111-114	
19215534-19	1991	The induction of c-fos by icv NMDA, and the partial blockade of light-induced c-fos expression by the antagonist MK801, are consistent with the hypothesis that glutamate mediates the effects of light on SCN activity.	Glutamic Acid	160-169	proto-oncogene c-Fos	Mesocricetus auratus	78-83